Anonymous ID: 617aca Oct. 10, 2021, 9 a.m. No.100107   🗄️.is 🔗kun   >>0108 >>0111

>>100103

top kek

 

Be Like: wtf these snowflakes sizzle

all pumped up on BS faschizzle

We do not want them in our homes

We'll wash them out with mighty tomes

We do not want them with our kids

We want them stowed away, with tight lids

We do not want them making decisions

They can't so much but sow divisions

It's time to bring this to a close

Q told how the story goes

We fight them day and night, we win

and hope upon hope for the suicide bin

Anonymous ID: 617aca Oct. 10, 2021, 3:16 p.m. No.100151   🗄️.is 🔗kun

>>100150

The binding of virions complexed with antibodies to Fcγ receptors on the target cells initiates receptor-mediated signaling events, leading to enhanced expression of inflammatory cytokines and suppression of intracellular antiviral responses at the transcriptome level, followed by endocytosis of the virus and subsequent activation of immune cells. The activated immune cells might accumulate in the lung and promote cytokine storm and lymphopenia.

 

also explains myocarditis as congestion, which prolongs inflammatory phase, retarding next phase of tissure healing

Anonymous ID: 617aca Oct. 10, 2021, 3:27 p.m. No.100162   🗄️.is 🔗kun

>>100156

in lay terms it's like this

 

  1. There is damage to tissue, trauma to vessels arteries, breaking of the skin etc

  2. Body response is "inflammation" which is a rushing of macrophages and matrix to clot or heal the wound, when the response is complete.....

  3. the tissue rebuilds with matrix sent

 

so what this article is saying is the process is arrested, kept in inflammatory stage, hence the tissue becomes congested with matrix and circulation slows to the area, lethal in the case of the heart and lungs, lethal in terms of clotting no matter where a the clots dislodge and move into the brain, heart, lungs