Anonymous ID: 2b13b2 Nov. 14, 2021, 12:29 a.m. No.108643   🗄️.is 🔗kun   >>8645 >>8698 >>8749 >>8758 >>8760

might come in handy....

 

°.~^Donna Carr^~»° @CarrHerth • 16h Replying to @DiscipleBearded

My friend went to the hospital a month ago with covid pneumonia, but he got monoclonal antibodies and was out within 6 hours 1-877- 332-6585 is the # he said they called to get pre-approved for monoclonal antibody treatment

 

https://twitter.com/CarrHerth/status/1459549077511852037

Anonymous ID: 2b13b2 Nov. 14, 2021, 12:48 a.m. No.108644   🗄️.is 🔗kun   >>8698 >>8749 >>8758 >>8760

FLUVOXAMINE FOR COVID INFLAMMATION

 

Investigating Antidepressants’ Surprising Effect on COVID Deaths

 

Researchers are still puzzling over what this drug does at the molecular level to help COVID patients

November 12, 2021

 

Researchers reported last month that an inexpensive, widely available pill substantially reduced hospitalizations and deaths in a large study of individuals with mild COVID symptoms who were at high risk for complications. It is the only existing oral medication with promising peer-reviewed data from multiple randomized COVID trials—and it is already used by millions of people worldwide. The drug is fluvoxamine, and it is approved in the U.S. for treating obsessive-compulsive disorder and depression. So how did this antidepressant end up in a trial for treating COVID?

 

“Drugs don’t know what their original indicated purpose was, they just do what they do, and they don’t usually do only one thing,” says Angela Reiersen, a child psychiatrist at Washington University in St. Louis (WashU). Along with her WashU psychiatry colleague Eric Lenze, Reiersen conducted a smaller randomized trial last year that suggested fluvoxamine could keep newly infected COVID patients from deteriorating.

 

Fluvoxamine is best understood for its impact on serotonin—a chemical messenger linked to mood and anxiety disorders. But the drug has other molecular targets. One is a protein called the sigma-1 receptor, which regulates the release of inflammatory molecules, including several that escalate in people with severe COVID. In a 2019 study, University of Virginia scientists chemically induced sepsis, a life-threatening infection complication, in mice. They observed that animals lacking the sigma-1 receptor developed severe inflammation, and many died. Yet in normal mice, a shot of fluvoxamine quieted the immunity overdrive and helped the animals survive.

 

That study caught Reiersen’s attention: the same overdrive immune response seen in those mice was a central feature observed in children who had come under her care because they had a rare genetic disorder—Wolfram syndrome—that led to psychiatric symptoms. When the pandemic hit and reports emerged that some people with COVID worsen because their inflammatory response goes haywire, Reiersen recalled the mouse study and wondered if fluvoxamine could also keep immune responses from spiraling out of control in these individuals.

 

https://www.scientificamerican.com/article/investigating-antidepressants-surprising-effect-on-covid-deaths/

Anonymous ID: 2b13b2 Nov. 14, 2021, 1:01 a.m. No.108646   🗄️.is 🔗kun   >>8647 >>8698 >>8749 >>8758 >>8760

Anti-inflammatory therapy for COVID-19 infection: the case for colchicine

 

Abstract

The search for effective COVID-19 management strategies continues to evolve. Current understanding of SARS-CoV-2 mechanisms suggests a central role for exaggerated activation of the innate immune system as an important contributor to COVID-19 adverse outcomes. The actions of colchicine, one of the oldest anti-inflammatory therapeutics, target multiple mechanisms associated with COVID-19 excessive inflammation. While many COVID-19 trials have sought to manipulate SARS-CoV-2 or dampen the inflammatory response once patients are hospitalised, few examine therapeutics to prevent the need for hospitalisation. Colchicine is easily administered, generally well tolerated and inexpensive, and holds particular promise to reduce the risk of hospitalisation and mortality due to COVID-19 in the outpatient setting. Successful outpatient treatment of COVID-19 could greatly reduce morbidity, mortality and the demand for rare or expensive care resources (front-line healthcare workers, hospital beds, ventilators, biological therapies), to the benefit of both resource-replete and resource-poor regions....

 

Keywords: chemokines; cytokines; immune system diseases; inflammatino; therapeutics.

 

https://pubmed.ncbi.nlm.nih.gov/33293273/

 

do your own research

available from alldaychemist.com

Anonymous ID: 2b13b2 Nov. 14, 2021, 1:15 a.m. No.108647   🗄️.is 🔗kun   >>8698 >>8749 >>8758 >>8760

>>108646

https://www.drugs.com/medical-answers/colchicine-treat-covid-19-coronavirus-3535211/

 

Can Colchicine be used to treat COVID-19 (coronavirus)?

 

https://www.drugs.com/medical-answers/colchicine-treat-covid-19-coronavirus-3535211/

 

It is an anti-inflammatory for gout that MAY calm down the cytokine storm seen sometimes with covid (and with vax reactions? not tested there afaik). Has been (and is still being) trialed with mixed results - some positive - but given the prejudice against any treatment except you-know-what, the results may be more positive than the attached article would imply.

Anonymous ID: 2b13b2 Nov. 14, 2021, 1:27 a.m. No.108648   🗄️.is 🔗kun

Because many MDs associated with large hospitals, universities and HMOs do not current treat Covid with anything other than the poisonous remdesivir, it's useful to know what else is being tried that is not yet well-known.

 

Also, the same remedies that defeat covid are likely to defeat jab after-effects, since they share in common the spike protein. Every day, more info is available, which can be posted both here and on the covid thread on qrbunker/endchan.

 

Each must do his own research but the more ordinary people know about what's going on in the way of genuine scientific research, the better.

Anonymous ID: 2b13b2 Nov. 14, 2021, 1:35 a.m. No.108653   🗄️.is 🔗kun   >>8655 >>8656

 

>>108652

idk, but i know that feeling of waking up feeling like sumtin's afoot

 

no big news, finally found another printer that's returnable locally (one of the challenges) - ordered it. Let's see if it stays in stock.

 

focusing a lot on covid stuff bc it is so pivotal in determining so much. turning that narrative around is a big deal, and it's on e of the few areas where there's new news almost every day. MDs and lawyers like Reiner F are pushing hard to gather the real facts and get them out to people

Anonymous ID: 2b13b2 Nov. 14, 2021, 1:41 a.m. No.108655   🗄️.is 🔗kun   >>8657 >>8680

>>108653

there is other news that matters, but the fear of covid has become the driving force behind the effort to gull people into accepting the great reset

 

>>108654

About one religion under God - that is an unusual thing to say. do you think he's saying Christianity or Judeo-Christian tradition?

Anonymous ID: 2b13b2 Nov. 14, 2021, 1:45 a.m. No.108658   🗄️.is 🔗kun

>>108654

I'ma thinking that people need to have the kind of sense that AA promoted, the sense of surrender to a higher power, in common. Not always a fan of religions per say, they can become corrupted as institutions. But the religious spirit - that's different.

Anonymous ID: 2b13b2 Nov. 14, 2021, 1:47 a.m. No.108659   🗄️.is 🔗kun

>>108656

had to give up on best buy, they have no stock. worse than other places, went with office depot. not gonna try to order something heavy and have to send it back in the mail if it's broke.

Anonymous ID: 2b13b2 Nov. 14, 2021, 1:49 a.m. No.108662   🗄️.is 🔗kun   >>8666

>>108660

i don't know about Sunday worship and the mark of the beast, never heard of that. what do you mean? that they will change the traditional meaning of Sunday to be its opposite?

Anonymous ID: 2b13b2 Nov. 14, 2021, 1:52 a.m. No.108664   🗄️.is 🔗kun

>>108661

the Left is definitely pushing a religion - the Green religion. First saw that in an issue of the APA Journal (amer psych assn) in 1992 - that early. psychologists saying that religion and science didn't have to conflict. next paragraph, they said coz it's the green religion. that was shocking at the time, green movement was new. thought they were crazy, would never sell.

 

did sell for a while, but green religion is past its prime. too many failed predictions. they might push it but not gonna go.

Anonymous ID: 2b13b2 Nov. 14, 2021, 2:01 a.m. No.108670   🗄️.is 🔗kun

>>108668

i'm interested but muh back is killin me, gotta go for now, will ck out stuff in the am. interesting name starting with "FLIP"

 

G'nite doc,

take care.

Anonymous ID: 2b13b2 Nov. 14, 2021, 9:35 a.m. No.108721   🗄️.is 🔗kun

>>108677

Hope for the best but not at all sure there will be a just outcome, if only bc the jury must be aware of the hue and cry of a mob. Judge now targeted too.

 

Re the blind listening to others:

if they did that, they wouldn't be blind.

willful blindness

Anonymous ID: 2b13b2 Nov. 14, 2021, 9:49 a.m. No.108733   🗄️.is 🔗kun

>>108680

>Flynn's idea of "one religion"

good points, all

religion relation to binding together - ligature

 

Bad guys always try to get their before - like with the idea of a One World Religion based on faulty values

A true one world 'religion' would be based on universal values that sustain life, the kind that transcend institutional doctrine.

 

I've got a hardcopy OED from around 1900. Not subject to instant editing, kek.

Anonymous ID: 2b13b2 Nov. 14, 2021, 10:02 a.m. No.108740   🗄️.is 🔗kun   >>8749 >>8758 >>8760

>>108725

HSBC and UBS AG:

both are creepy

HSBC was established to support the opium trade in China and UBS AG is implicated is stealing and still retaining Jewish gold during WW2. Blood money, both - and right there in wikipedia.

 

Take a look at the reconstructed election fraud thread on qrbunker - look at both hsbc and ubs ag. Totally committed to globalist agenda, and they are not shy about stating it publicly.

 

HSCB owns DOMINION patents and UBS AG owns a chunk of DOMINION via ubs llc in nyc. (u know this but other readers might not).

 

link to start of WHO OWNS DOMINION discussion. It's a long one:

https://endchan.org/qrbunker/res/9922.html#10508