Anonymous ID: a4e8fe Dec. 2, 2020, 11:11 p.m. No.11884283   🗄️.is 🔗kun   >>4297 >>4386

Explanation for high death rates in minorities with WuFlu

 

Nonwhite men with G6PD genetic deficiency are very susceptible to severe reactions to certain medicines in coronavirus treatment

 

This blog entry of picrel appears in the middle of the website shown below:

https://europa.is/global/coronacrisis/risky-vaccination-experiments-dr-wodarg-and-dr-yeadon-eu-petition/

https://archive.md/JBzuE

 

The high death rates are due to "glucose-6-dehydrogenase deficiency, or 'G6PD deficiency', one of the most common genetic peculiarities, which can lead to threatening haemolysis (dissolution of red blood cells), mainly in men, when certain drugs or chemicals are taken," especially hydroxychloroquine and acetylsalicylic acid (aspirin).

 

German pulmonologist and former member of the German Parliament Dr. med. Wolfgang Wodarg writes that, "The G6PD deficiency then leads to a disruption of the biochemical processes in the red blood cells and – depending on the dose – to mild to life-threatening haemolysis. The debris of the burst erythrocytes subsequently leads to microemboli, which block small vessels throughout the organs." … "This enzyme deficiency "occurs in many regions of Africa in 20 – 30% of the population. It is … one of the most common genetic peculiarities, which can lead to threatening haemolysis (dissolution of red blood cells), mainly in men, when certain drugs or chemicals are taken."

 

"This hereditary trait is particularly common among ethnic groups living in areas with malaria. The modified G6PD gene offers advantages in the tropics. It makes its carriers resistant to malaria pathogens. However, G6PD deficiency is also dangerous if those affected come into contact with certain substances found in, for example, field beans, currants, peas and a number of medicines. These include acetylsalicylic acid, metamizole, sulfonamides, vitamin K, naphthalene, aniline, malaria drugs and nitrofurans."

 

Most studies don't take the G6PD deficiency into account when studying HCQ and coronavirus: "There are now hundreds of trials worldwide, planned or ongoing by different sponsors, in which HCQ is used alone or together with other drugs. When I looked at some large studies to see if patients with G6PD deficiency were excluded, I found no evidence of this in most study plans. In the USA, for example, a large multi-center study with 4,000 volunteers from healthy medical staff is being prepared. Here, however, the term “hypersensitivity” is only used in general terms, as is the case with all drugs with regard to allergic reactions. In a chloroquine/hydroxychloroquine study by Oxford University (NCT04303507) with a planned 40,000 participants, the risk of G6PD deficiency is also not mentioned. In another large study by the Pentagon, though, there is an explicit warning to exclude G6PD deficiency patients from the study."

Anonymous ID: a4e8fe Dec. 2, 2020, 11:32 p.m. No.11884386   🗄️.is 🔗kun

>>11884283

>>11884297

 

Wow! G6PD also explains the high rates of hypertension and diabetes mellitus (type 2 diabetes) in blacks!

 

G6PD is glucose-6-phosphate dehydrogenase deficiency, a genetic defect present from birth. If I understand correctly, the normal gene protects against malaria.

 

https://pubmed.ncbi.nlm.nih.gov/11763298/

 

Ethn Dis - Fall 2001;11(4):749-54.

G6PD deficiency: its role in the high prevalence of hypertension and diabetes mellitus

R S Gaskin 1, D Estwick, R Peddi

PMID: 11763298

 

Abstract

Hypertension and diabetes mellitus represent increasing threats to the health of many populations. For reasons not completely understood, the prevalence of these diseases is higher in some ethnic groups than in others. The key to this puzzle may rest with the interplay of a defect of an enzyme-mediated process and the environment. Oxidative stress and impairment of synthesis or release of nitric oxide (NO) are being regarded as causative factors in the pathogenesis of hypertension, diabetes mellitus and atherosclerosis, among other conditions. Glucose-6-phosphate dehydrogenase (G6PD) deficiency has been overlooked as a cause of both oxidative stress and a decrease in the generation of nitric oxide (NO). G6PD generates nicotinamide adenine dinucleotide phosphate (NADPH), a co-factor in the synthesis of nitric oxide. There is impairment of the production of nitric oxide superoxide and hydrogen peroxide in G6PD-deficient granulocytes. In the polyol pathway, G6PD deficiency causes hyperglycemia, making more glucose available for the non-enzymatic production of advanced glycosylation end products (AGE's), which also causes an increase in superoxide anions and a quenching of nitric oxide. Currently, there are 200 million people worldwide with red cell x-linked chromosome defects who, with the persistent ingestion of refined carbohydrates, are at greater risk of developing hypertension or diabetes mellitus than those racial groups without the defect.

Anonymous ID: a4e8fe Dec. 3, 2020, 12:14 a.m. No.11884610   🗄️.is 🔗kun

>>11884379

 

https://www.biblegateway.com/

 

Good resource to read various versions of the Bible online. Can read entire books, chapters, or verses. Very versatile with around 60 different English translations.