Anonymous ID: d68a0b Jan. 24, 2021, 9:16 a.m. No.12696829   ๐Ÿ—„๏ธ.is ๐Ÿ”—kun   >>6875

The rod itself would penetrate hundreds of feet into the Earth, destroying any potential hardened bunkers or secret underground sites. More than that, when the rod hits, the explosion would be on par with the magnitude of a ground-penetrating nuclear weapon โ€“ but with no fallout.

 

It would take 15 minutes to destroy a target with such a weapon.

 

https://www.wearethemighty.com/articles/these-air-force-rods-from-god-could-hit-with-the-force-of-a-nuclear-weapon/

Anonymous ID: d68a0b Jan. 24, 2021, 9:20 a.m. No.12696859   ๐Ÿ—„๏ธ.is ๐Ÿ”—kun

A new study in mice by researchers from the Francis Crick Institute in London, United Kingdom, now suggests that antibiotics could actually also โ€œprimeโ€ the lungs for viral infections.

 

The researchersโ€™ findings, which feature in the journal Cell Reports, also show that gut bacteria drive a type of protein signaling that helps the cells that line the lungs keep the flu virus from spreading. Antibiotic use, it seems, interferes with this protein signaling and thus impairs this first line of defense.

 

โ€œWe found that antibiotics can wipe out early flu resistance, adding further evidence that they should not be taken or prescribed lightly,โ€ explains lead researcher Andreas Wack, Ph.D.

 

The team noticed that approximately 80% of the untreated mice with healthy gut bacteria survived the infection with the flu virus. Yet, of the mice who had previously received the antibiotic mix, only one-third were able to survive the viral infection.

 

โ€œInappropriate use [of antibiotics] not only promotes antibiotic resistance and kills helpful gut bacteria, but may also leave us more vulnerable to viruses,โ€ says Wack.

 

โ€œWe were surprised to discover that the cells lining the lung, rather than immune cells, were responsible for early flu resistance induced by microbiota,โ€ notes Wack.

 

The process by which antibiotics seem to render the lungs more vulnerable to viral infections is a complex one, and it relates, in part, to when and how the immune response occurs.

 

Gut bacteria usually send interferon signals that switch on the antiviral gene Mx1 in mice, corresponding to a similar gene called MxA in humans. However, antibiotic treatment delays the switching on of the antiviral gene, affecting the efficiency of the response that the body initiates against the virus.

 

โ€œIt takes around 2 days for immune cells to mount a response, in which time the virus is multiplying in the lung lining,โ€ explains Wack.

 

โ€œTwo days after infection, antibiotic-treated mice had five times more virus in their lungs. To face this bigger threat, the immune response is much stronger and more damaging, leading to more severe symptoms and worse outcomes,โ€ he continues.

 

https://www.medicalnewstoday.com/articles/325651#The-defensive-role-of-gut-bacteria