'Luc Montagnier HIV Nobel Prize HIV Virologist Says CV "Bio-engineered" in Lab - Contains Strings Of Inserted HIV

"This virus was manipulated meticulously, it's not natural."

Interesting Points:

-Mutation-Deletions are bringing the virus back to its unmodified state - the HIV insertion is specifically degrading faster than the rest.

-The virus is quickly loosing its ability to infect humans.

-Verifiable proof this was engineered "For Gain-of-function" advances, specifically contains a genetic sequence from HIV.

-China has enormous pressure on any studies or media related to the origins of the virus.

-Source as the Wuhan BioLab confirmed.

-Intentional or not, this engineered virus being released constitutes as a launched Bio Weapon.

-Electromagnetic means of manipulation can be used to neutralize the virus. Which also means they can be used to amplify it.

-Who controls the modulation of the antennas determines the effect

Host: "If we want to reassure ourselves, since you have continued to work on this virus, that virus today it has lost, you say, all its virulence and you imagine that it will no longer be specifically dangerous in the next few weeks?"

Luc: "Yes, what is interesting, if you will, is that my mathematical colleague Perez thinks there's some kind of harmonization of the sequences, of the genetic information, which is carried as well in our own chromosomes as by the virus. So nature doesn't accept anything. That is, we can do anything to nature but if you're making an artificial construct, it is unlikely to survive. That is to say that nature loves the harmonious things and what is foreign, such as another virus coming from another virus is not well tolerated. So, what is happening as time goes by, the epidemic is spreading, notably in the United States where there is the largest number of cases, there is an evolution through mutations, right. The sequences will mutate. Mutate means one nucleotide changed for an other, the genetic code is changing. But it also means deletions that are made: It means that there are whole chunks of the genome, including… what is extraordinary is that, precisely, the region that carries the HIV sequences are mutating much faster than the others. So, it itself disappears by deletions. So, we have patients on whom we have isolated the virus and sequenced the virus, in the western part of the United States, in Seattle, and this sequence is almost demolished and no longer works."

Host: "So the HIV is gone?"

Luc: "You'd think if the new pathogenicity if you will, of the corona is related to the introduction of these sequences, that it is going to disappear. It's a bit of hope."

https://www.youtube.com/watch?v=durcHyxpFT4

>"It was like an aggressive tiger in March and April but now it's like a wild cat," Bassetti said. "Even elderly patients, aged 80 or 90, are now sitting up in bed and they are breathing without help. The same patients would have died in two or three days before."

https://www.theblaze.com/news/coronavirus-has-significantly-weakened-could-disappear-without-vaccine-top-doctor-reveals

>Evidence continues to mount that the new infections throughout much of the country are extremely mild, with the exception of some serious cases coming over the border from Mexico.

https://www.conservativereview.com/news/horowitz-evidence-coronavirus-cases-now-weaker-flu/

"WHO report finds coronavirus probably emerged in bats, 'extremely unlikely' to be result of lab leak"

NOPE

A Bayesian analysis concludes ''beyond a reasonable doubt that SARS-CoV-2 is not a natural zoonosis but instead is laboratory derived''

https://archive.is/eOJsU

''Evidence: Lack of seroconversion in Wuhan and Shanghai.''

"A hallmark of zoonotic infections (vertebrate animal host-to-human microbial infection) is repeated, abortive jumps into humans over time until sufficient ‘human-adapted’ mutations permit efficient human-to-human spread and further evolution"

''Evidence: Lack of posterior diversity for SARS-CoV-2 compared to MERS and SARS-CoV-1''

The earliest stages of human CoV-1 and MERS infections were characterized by viral genome base diversity as expected for multiple, independent jumps from a large and diverse intermediate host population into humans.

Combining MERS and CoV-1 studies, out of the earliest 255 human infections in which virus genome sequences are available, 137 could not be rooted in a prior human-to-human infection and so are attributed to an independent intermediate host-to-human infection.

That is about 54% non-human-to -human transmission.

On the other hand, Ralph Baric has written68 that CoV-2 is different: “SARS-CoV-2 probably emerged from bats, and early strains identified in Wuhan, China, showed limited genetic diversity, which suggests that the virus may have been introduced from a single source.” [emphasis added.]

With CoV-2, there are 249 viral genomes in GISAID from Hubei province, where Wuhan is located, collected between Dec 24, 2019 and Mar 29, 2020.

From Dec 24, 2019 to November 2020, there are 1001 genomes sequenced from all of China and 198,862 worldwide.

For CoV-2, every single genome sequence is rooted in the first sequence from the PLA Hospital in Wuhan.

Not one case of posterior diversity.

''Evidence: Opportunity.''

The Wuhan Institute of Virology has publicly disclosed that by 2017 it had developed the techniques to collect novel coronaviruses, systematically modify the receptor binding domain to improve binding or alter zoonotic tropism and transmission, insert a furin site to permit human cell infection, make chimera and synthetic viruses, perform experiments in humanized mice, and optimize the ORF8 gene to increase human cell death (apoptosis).

''Evidence and Motive for laboratory furin site insertion:''

A key to infectivity of coronaviruses is the addition, in nature or the laboratory, of a furin cleavage site (FCS) at the S1/S2 junction of the Spike Protein. Furin cleavage sites (FCS) have been widely understood to be important for many viral infections, including HIV, influenza, and others. It has also been widely understood before now that lineage B coronaviruses do not have FCS.It was therefore surprising when an examination of SARS-CoV-2 Spike Protein found an insertion of a 12-nt, 4-AA sequence near the junction of the S1/S2 subunits which creates a furin site that is essential to human infectivity and transmission. As expected from previous work, no lineage B (sarbecovirus) coronavirus has this feature. This is the most difficult “molecular fingerprint” of SARS-CoV-2 to explain having been acquired in the wild and for that reason there are no even passingly feasible theories. One database of whole genome sequences of 386 coronaviruses was devoid of furin cleavage sites.78 Another database of 2956 genomes of sarbecovirus strains sequences shows that none have a furin site.79 This is a highly significant finding with a probability that sarbecovirus has a furin site in the wild of one in about 985.

''Evidence: Codon usage can distinguish insertion events in the wild from those created in the laboratory.''

Decades of research has identified that all life forms, viruses, bacteria, and humans alike, use the codons in a signature pattern of frequency which can be used to identify a particular sequence of RNA or DNA as human or non-human; viral or non-viral.In this way, viruses in nature and scientists in the laboratory, with different goals and motivations, make distinguishing codon usage decisions which can sometimes provide a fingerprint of their source.

''Evidence. Laboratory codon optimization uses CGG for laboratory insertions of arginine residues 50% of the time.''

''Evidence: SARS-CoV-2 Spike Protein is Highly Optimized for ACE2 Binding and Human Cell Infectivity, a Finding that is Inconsistent with Natural Selection but is Consistent with Laboratory Creation''