Now more than ever do we see the dangers of Spike Protein Therapy!
OVERWHELMING EVIDENCE COVID-19 IS AN ONCOGENIC, HYPERINFLAMMATORY MITOCHONDRIAL DISEASE OF IRON METABOLISM CAUSED BY THE SPIKE PROTEIN, DISTINCTLY SEPARATE FROM THE ACCOMPANYING MILD RESPIRATORY VIRUS.
A paper published on medRxiv yesterday shows that a urine biomarker is a key indicator of kidney injury and death in COVID-19. The marker identified is NGAL. This marker is usually used for diagnosis in TRAUMATIC INJURY PATIENTS as it shows IMMEDIATE damage. Yet, what is NGAL?
NGAL is neutrophil gelatinase-associated lipocalin, or Lipocalin-2. What does this protein do, and why is it expressed? This protein is part of the INNATE IMMUNE SYSTEM and is expressed in conjunction with a BACTERIAL infection to SEQUESTER IRON, preventing its use by the invading organism.
Perhaps the most important paper yet published (referenced) about COVID-19 was published last summer to little fanfare. In it, the authors note that the SPIKE PROTEIN has a MUCH HIGHER AFFINITY for PORPHYRIN MOLECULES than ACE2. At the same time the virus increases free heme and free iron (Fe), but more importantly, decreases functional hemoprotein. COVID-19 produces dysfunctional hemoproteins and dysfunctional porphyrin that are no longer capable of making heme. This leads to more hemoprotein available for COVID-19 to bind to, which leads to the release of more free iron, and as a result, increased inflammation [6]. In addition, the iron released by dying cells has additive toxic effects.
This has devastating consequences on the mitochondria. The ROS formed by COVID-19′s iron-induced “cytokine storm” create inflammation that is destructive to the mitochondria in the electron transport chain (ETC). If insufficient mitochondria in cells is evident, such as in white adipose cells, these cells are unable to accommodate the severe ROS formed leading to overwhelming inflammation. Brown adipose cells are better at handling ROS due to higher concentrations of mitochondria.
This explains why the OBESE (and even those that are not obese and have a "sweet tooth!") are at much higher risk. Their fat cells HAVE FEWER MITOCHONDRIA TO DEAL WITH THE METABOLIC STRESS! They have an overwhelming ENERGY DEFICITY and CANNOT CONTINUE THE METABOLIC BATTLE. They collapse due to "battle fatigue" and die.
And if you survive? The unprecedented outpouring of toxic iron into the body is most likely the cause of Long COVID and the incoming cancers and neurodegenerative diseases. Excess iron is tightly associated with tumorigenesis in multiple human cancer types through a variety of mechanisms including catalyzing the formation of mutagenic hydroxyl radicals, regulating DNA replication, repair and cell cycle progression, affecting signal transduction in cancer cells, and acting as an essential nutrient for proliferating tumor cells.
Please understand the major finding. All of this is due to the SPIKE PROTEIN's interactions. COVID-19 produces a severe systemic inflammatory reaction, likely the result of increased free heme levels and decreased levels of HO-1 activity and functional hemoprotein. The spike protein of COVID-19 binds ACE-2 receptors and porphyrin molecules with weak and strong affinity, respectively. Porphyrins are the building blocks of heme and allow COVID-19 access to invade cells along with ACE-2 receptors and bind functional hemoprotein within the cell.
Now, more than ever, do we see the dangers of spike therapies. IVM ACCELERATES PORPHYRIN CLEARANCE!!!!!
Study: https://www.medrxiv.org/content/10.1101/2021.06.10.21258638v1