Anonymous ID: 168df7 July 31, 2021, 3:49 p.m. No.14239765   🗄️.is 🔗kun   >>9782 >>9799 >>9831

1,25(OH)2D3 inhibition.

Decreases CYP27B1 which aids in 1-alpha-hydroxylase production to create 1,25(OH)2D3.

Increases CYP24A1 which produces 24-hydroxylase which causes overuse of available 1,25(OH)2D3. It also breaks down 25-hydroxyvitamin which is the form of Vit-D which is stored in the fat.

>high fructose corn syrup causes Vit D deficiency

>which leads to vulnerability to covid

>no fast food places closed during the pandemic

Anonymous ID: 168df7 July 31, 2021, 3:53 p.m. No.14239782   🗄️.is 🔗kun   >>9811 >>9831 >>0224 >>0239 >>0246 >>0255 >>0265 >>0309

>>14239765

Consumers of high levels of fructose/sucrose are unable to create more usable vitamin D, even with supplementation, sun exposure, and are burning their stored vit D via a series of oxidative stress reactions

 

"ROS generated by fructosylation increases oxidative damage and stress response in the inner of cell, leading to DNA damage and proinflammatory cytokines production."

 

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5409744/

Anonymous ID: 168df7 July 31, 2021, 3:59 p.m. No.14239811   🗄️.is 🔗kun   >>9831 >>0265

>>14239799

LETS FUCKING GOOOOO

>>14239782 (continued)

Consider a couple things.

high % of "covid" deaths had elevated BMI

HFCS changes the gut biome which has been found to interfere with Ca+ transport which interferes with raw absorption of Vit D rich foods.

HFCS contributes to NAFLD which damages cells in the liver responsible for the production of 25-hydroxylase.

Oxidative stress on the kidneys, plus elevated blood sugar levels (with high BMI, diabetes) causes a reduction in 1-alpha-hydroxylase.

 

This is hypothesized as a link to why people without diabetes or liver disease died from acute liver and kidney failure. (refer to PMCID: PMC7515790)

Anonymous ID: 168df7 July 31, 2021, 4:02 p.m. No.14239831   🗄️.is 🔗kun   >>9837 >>9912 >>0024 >>0265 >>0279 >>0312

in conclusion

>>14239811

>>14239765

>>14239782

>>14239799

Sars Cov-2 leads to decreased ACE2

Increasing AT-II, decreasing AT 1,7 (angiotensin)

Increasing super oxide

The oxidative stress causes a dysfunction with endothelial cells, these are the cells that cover the vasculature.

Increasing VWF (Von willebrands factor)

Leading to the thrombosis.

Referring to NEJM report from May 2020 that Sars Cov-2 patients had 9 times the amount of clots in their lungs than the control groups with influenza virus in their lungs. Subsequent studies in the last year have demonstrated similar findings with liver failure and kidney failure with blood clots found during autopsy.

 

HIGH FRUCTOSE CORN SYRUP IS ANTI-CHRIST