Xenobots are here: Tiny bio-robots inside your veins may heal you & will definitely be weaponized by West to take out ‘bad guys’
18 Jan, 2020
https://www.rt.com/news/478530-biological-robots-designed-by-computer/
WHAT !? WAIT THAT'S CONTRAINDICATED TREATMENT IN PROSTATE CANCER PATIENTS! THE ANTI ANDROGEN'S ( Estrogen) ARE CAUSING IT.
https://www.telegraph.co.uk/science/2016/11/30/man-cured-prostate-cancer-doctors-shock-tumour-death-testosterone/
Man 'cured' of prostate cancer after doctors shock tumour to death with testosterone.
https://www.testosterone.me/testosterone-injection-prostate-cancer-treatment
https://www.science.org/content/article/cancer-paradox-testosterone-injections-combat-lethal-prostate-tumors
https://medicalxpress.com/news/2019-03-testosterone-prostate-cancer-recurrence-low-risk.html
https://naturalon.com/can-boosting-testosterone-actually-fight-prostate-cancer/view-all/
more;
https://theantimedia.com/cancer-vaccine-kills-tumors/
Reasons why Testosterone kills prostate cancer ….
Rational :Hypothesis (Note: an MD)
Is it possible that by depriving the prostate cells from androgens allows an unnatural balance of estrogen causing cancer ?
Estrogen receptors (ERα and or ERβ) in certain prostate cells are now receiving too much unregulated estrogen data. These molecular signals are now trying to convince the prostate tissue under estrogen dominance to become uterine tissue. Due to the lack of genetic DNA available to become totally uterine tissue, cell division runs short of the process creating bastard cells (cancer), neither prostatic nor uterine but still displaying component DNA of a Y chromosome. It would be interesting to see the estrogen levels of patients with prostate cancer in relation to testosterone prior to standard treatment during and after .
.Another fact is "T" metabolizes into both estrogen and DHT in the prostate. How this may be affecting the results would have to be studied, I'm sure its out there. Some studies have shown that administration of DHT reduces PSA levels. Could this be due to additional 3β-Adiol produce via 3β-HSD ( see chart) etc. and DHT supplementation?
There is a possibly of a natural process of limited rate but constant and controlled apoptosis in the prostate like its female analog (uterus ) that occurs but for some reason it is reduced or fails to occur. This would be a different process than basic cellular atrophy i.e. death.
-
Male estrogen dominance due to age or over conversion increases additional ERβ in prostate cells and the latent effects in cell divisions via access through ERα.
-
Increased ERβ aids in additional estrogen uptake creating malignancy.
-
3β-Adiol over stimulates ERβ resulting in apoptosis due to limited estrogen supply.
The 3β-Adiol stimulates ERβ is interesting in that it causes cell death in the prostate much like the uterus is affected during a cycle. This death my be occurring because the stimulation of the ERβ via 3β-Adiol without estrogen present.
In other words increase 3β-Adiol to max amounts stimulating the ERβ while reducing and or removing estrogen ( anticipation leads to devastation , think drug withdrawal) The higher the climb the harder the fall (apoptosis).
Stimulating the ERβ to a higher than normal threshold, then starving it. Cell death is an occurrence of increasing the need while removing the sustenance via some mechanism. This apoptosis in turn reducing the number of abnormal cell divisions of those bastard cells.
3β-Adiol is created from 5α-DHT, catalyzed by the enzyme 3β-hydroxysteroid dehydrogenase (3β-HSD). 3β-Adiol does not bind to androgen receptors; rather 3β-Adiol is a potent stimulator of Estrogen Receptor-β (ERβ). In the prostate, 3β-Adiol’s stimulation of ERβ prevents excess cell proliferation and stimulates apoptosis and re-differentiation.
testosterone metabolism
This could be contrary to some treatments of estrogen injections for prostate cancer in that the treatments are causing more harm than good.
High "T" dosage, DHT, 3α-HSD,17β-HSD, 3β-HSD, with 3β-Adiol
combined with an anti aromatase (ARIMIDEX) and estrogen blocker(Tomoxifin)
"T" may not be the culprit but either too much estrogen with an imbalance of DHT, 3α-HSD,17β-HSD, 3β-HSD and 3β-Adiol