Anonymous ID: 9d5214 Nov. 19, 2021, 6:18 p.m. No.15040076   ๐Ÿ—„๏ธ.is ๐Ÿ”—kun   >>0202 >>0240 >>0242 >>0456 >>0516 >>0548 >>0629

>>15039973

Anons! Everything is parasites- seriously

 

https://www.pbs.org/wgbh/nova/insidenova/2010/06/t-gondii.html

 

Here's how the bug works: Humans ingest Toxoplasma gondii, a common relative of malaria, which, at first, only makes itself known through mild flu symptoms. These symptoms have been known to linger in individuals with compromised immune systems, such as those affected by AIDS, but, in the majority of cases, the symptoms pass, the host feels better, and the bug is forgotten.

 

But Toxoplasma gondii stays onโ€ฆ

 

Dormant in the host's brain, it forms cysts. Research suggests that the parasite begins to affect how the brain responds to dopamine, a hormone that works as a neurotransmitter. And, gradually, it changes the host's personality.

 

Scientists have recorded four ways in which T. gondii affects the host. First, individuals with the parasite are more likely to experience guilty feelings, classified in studies as a type of neuroticism, than those who are not infected.

 

Second, researchers have found that the parasite's human hosts are more likely to be uncoordinated. One study demonstrated that both drivers and pedestrians who have been in traffic accidents are three times more likely to have been exposed to T. gondii.

 

Third, those who are infected with T. gondii are less likely to seek out new experiences. In researchers' words, they are less prone to "novelty seeking" and are more likely to demonstrate characteristics of "uncertainty avoidance."

 

And fourth, and perhaps most frighteningly, the presence of Toxoplasma gondii in human hosts shows a correlation with schizophrenia. People who suffer from schizophrenia are, in fact, three times more likely to carry T. gondii than those who do not. While unsettling, the correlation is not altogether surprising; schizophrenia is related to abnormalities in the way the brain receives dopamine, the hormone that Toxoplasma gondii also affects.

 

Scientists have observed other trends as well, including skewing sex ratios so that in heavily-affected populations, more males are born.

 

So what is Toxoplasma gondii, and what does it want with us? Most often described as a "cat parasite," the bug has three hosts: humans, cats, and rodents.

 

In cats, the parasite lives in the wall of the small intestine and is spread through the cats' feces. Then other animals ingest it, including rodents and humans; in both, it forms cysts in the brain, liver and muscle tissues.

 

The brain cysts affect the rodents even more dramatically than they affect humans. Research conducted by Joanne Webster at Imperial College, London seems to indicate that T. gondii causes rodents to draw predators' attention by wandering in the open; in some cases, it seems that they may even seek out the smell of cats. Of course, if a cat eats the rodent, Toxoplasma gondii's lifecycle starts over within the cat's intestines, so it's in the bug's best interest to hijack these rodents.

 

Interestingly, the dopamine-blocking Haloperidol, the drug used to treat schizophrenia in humans, reverses the fatal effects of T. gondii in rodents.

 

Over the last five years or so, evidence has been building that some human cultural shifts might be influenced, or even caused, by the spread of Toxoplasma gondii. Kevin Lafferty, the author of the USGS study, said in the September 2006 issue of soundwaves, the USGS newsletter, "The geographic variation in the latent prevalence of Toxoplasma gondii may explain a substantial proportion of human population differences we see in cultural aspects that relate to ego, money, material possessions, work, and rules."

 

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