Anonymous ID: 508748 Dec. 23, 2021, 7:34 a.m. No.15242466   🗄️.is 🔗kun   >>2483 >>2525 >>2553 >>2673

WHY [THEY] HATE IVERMECTIN

 

Not only does it blow-up their fear pandemic

 

IT ALSO CURES CANCER!

 

Esophageal, Colorectal and Breast Cancer Just (3) medical studies in 2021. Just the tippy top of a HUUUGE cancer therapeutic iceberg!

 

1) "Ivermectin induces apoptosis of esophageal squamous cell carcinoma via mitochondrial pathway"

BMC Cancer volume 21, Article number: 130

Published: 07 December 2021

 

Abstract - Background

Esophageal squamous cell carcinoma (ESCC) is the most predominant primary malignant tumor among worldwide, especially in China. To date, the successful treatment remains a mainly clinical challenge, it is imperative to develop successful therapeutic agents.

 

Methods

The anti-proliferative effect of ivermectin on ESCC is investigated in cell model and in nude mice model. Cell apoptosis was assessed using flow cytometry, TUNEL assay and western blotting. Mitochondrial dysfunction was determined by reactive oxygen species accumulation, mitochondrial membrane potential and ATP levels.

 

Results

Our results determined that ivermectin significantly inhibited the proliferation of ESCC cells in vitro and in vivo. Furthermore, we found that ivermectin markedly mediated mitochondrial dysfunction and induced apoptosis of ESCC cells, which indicated the anti-proliferative effect of ivermectin on ESCC cells was implicated in mitochondrial apoptotic pathway. Mechanistically, ivermectin significantly triggered ROS accumulation and inhibited the activation of NF-κB signaling pathway and increased the ratio of Bax/Bcl-2.

 

Conclusions

These findings indicated that ivermectin has significant anti-tumor potential for ESSC and may be a potential therapeutic candidate against ESCC.

 

https://bmccancer.biomedcentral.com/articles/10.1186/s12885-021-09021-x

 

2) Front. Pharmacol., 13 August 2021 | https://doi.org/10.3389/fphar.2021.717529

 

"Ivermectin has New Application in Inhibiting Colorectal Cancer Cell Growth"

 

Infection and Immunity Institute and Translational Medical Center of Huaihe Hospital, Henan University, Kaifeng, China

 

Colorectal cancer (CRC) is the third most common cancer worldwide and still lacks effective therapy. Ivermectin, an anti-parasitic drug, has been shown to possess anti-inflammation, anti-virus, and antitumor properties. However, whether ivermectin affects CRC is still unclear. The objective of this study was to evaluate the influence of ivermectin on CRC using CRC cell lines SW480 and SW1116. We used CCK-8 assay to determine the cell viability, used an optical microscope to measure cell morphology, used Annexin V-FITC/7-AAD kit to determine cell apoptosis, used Caspase 3/7 Activity Apoptosis Assay Kit to evaluate Caspase 3/7 activity, used Western blot to determine apoptosis-associated protein expression, and used flow cytometry and fluorescence microscope to determine the reactive oxygen species (ROS) levels and cell cycle. The results demonstrated that ivermectin dose-dependently inhibited colorectal cancer SW480 and SW1116 cell growth, followed by promoting cell apoptosis and increasing Caspase-3/7 activity. Besides, ivermectin upregulated the expression of proapoptotic proteins Bax and cleaved PARP and downregulated antiapoptotic protein Bcl-2. Mechanism analysis showed that ivermectin promoted both total and mitochondrial ROS production in a dose-dependent manner, which could be eliminated by administering N-acetyl-l-cysteine (NAC) in CRC cells. Following NAC treatment, the inhibition of cell growth induced by ivermectin was reversed. Finally, IVERMECTIN at low doses (2.5 and 5 µM) INDUCED CRC CELL ARREST. Overall, IVERMECTIN SUPPRESSED CELL PROLIFERATION by promoting ROS-mediated mitochondrial apoptosis pathway and inducing S phase arrest in CRC cells, SUGGESTING THAT IVERMECTIN MIGHT BE A NEW POTENTIAL ANTICANCER DRUG THERAPY FOR HUMAN COLORECTAL CANCER AND (((OTHER CANCERS))).

 

https://www.frontiersin.org/articles/10.3389/fphar.2021.717529/full

 

3) Use of the Anti-Parasitic Drug Ivermectin to Treat Breast Cancer

 

Lee, Peter P. MD Oncology Times: May 5, 2021 - Volume 43 - Issue 9 - p 10

 

OUR GROUP FOUND THAT IVERMECTIN, AN ANTI-PARASITIC DRUG USED WORLDWIDE SINCE 1975 TO TREAT CLOSE TO 1 BILLION PEOPLE PRIMARILY FOR RIVER BLINDNESS AND OTHER PARASITIC INFECTIONS, PROMOTES ICD IN BREAST CANCER CELLS. Our findings on this novel therapeutic combination published recently in npj Breast Cancer journal (2021; https://doi.org/10.1038/s41523-021-00229-5). THIS IS THE FIRST TIME A RESEARCH TEAM HAS DEMONSTRATED THAT CHECKPOINT INHIBITORS CAN BE USED TO SUCCESSFULLY TREAT BREAST CANCER - WHEN COMBINED WITH IVERMECTIN, AN INEXPENSIVE, EXISTING SAFE DRUG. Ivermectin is safe and inexpensive at roughly $30 a dose, making it attainable for everyone including cancer patients in developing countries.

 

https://journals.lww.com/oncology-times/fulltext/2021/05050/use_of_the_anti_parasitic_drug_ivermectin_to_treat.4.aspx 

Anonymous ID: 508748 Dec. 23, 2021, 7:49 a.m. No.15242553   🗄️.is 🔗kun   >>2571 >>2588 >>2673

>>15242466

Anons, how long before PubMed Scrubs

 

All Studies Ivermectin?

 

"Ivermectin, a potential anticancer drug derived from an anti-parasitic drug"

 

Pharmacol Res. 2021 Jan; 163: 105207.

Published online 2020 Sep 21. doi: 10.1016/j.phrs.2020.105207

 

Graphical abstract

Ivermectin has powerful antitumor effects, including the inhibition of proliferation, metastasis, and angiogenic activity, in a variety of cancer cells. This may be related to the regulation of multiple signaling pathways by ivermectin through PAK1 kinase. On the other hand, ivermectin promotes programmed cancer cell death, including apoptosis, autophagy and proptosis. Ivermectin induces apoptosis and autophagy is mutually regulated. Interestingly, ivermectin can also inhibit tumor stem cells and reverse multidrug resistance and exerts the optimal effect when used in combination with other chemotherapy drugs.

 

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7505114/

 

"Ivermectin: enigmatic multifaceted 'wonder' drug continues to surprise and exceed expectations"

 

Review J Antibiot (Tokyo)

2017 May;70(5):495-505. doi: 10.1038/ja.2017.11. Epub 2017 Feb 15.

 

Abstract

Over the past decade, the global scientific community have begun to recognize the unmatched value of an extraordinary drug, ivermectin, that originates from a single microbe unearthed from soil in Japan. Work on ivermectin has seen its discoverer, Satoshi Ōmura, of Tokyo's prestigious Kitasato Institute, receive the 2014 Gairdner Global Health Award and the 2015 Nobel Prize in Physiology or Medicine, which he shared with a collaborating partner in the discovery and development of the drug, William Campbell of Merck & Co. Incorporated. Today, ivermectin is continuing to surprise and excite scientists, offering more and more promise to help improve global public health by treating a diverse range of diseases, with its unexpected potential as an antibacterial, antiviral and anti-cancer agent being particularly extraordinary.

 

https://pubmed.ncbi.nlm.nih.gov/28196978/

Anonymous ID: 508748 Dec. 23, 2021, 7:56 a.m. No.15242588   🗄️.is 🔗kun

>>15242553

BRAIN CANCER !!

 

"Ivermectin inhibits the growth of glioma cells by inducing cell cycle arrest and apoptosis in vitro and in vivo"

 

J Cell Biochem

2019 Jan;120(1):622-633. doi: 10.1002/jcb.27420. Epub 2018 Sep 14.

 

Abstract

Glioma, the most predominant primary malignant brain tumor, remains uncured due to the absence of effective treatments. Hence, it is imperative to develop successful therapeutic agents. This study aimed to explore the antitumor effects and mechanisms of ivermectin (IVM) in glioma cells in vitro and in vivo. The effects of IVM on cell viability, cell cycle arrest, apoptosis rate, and morphological characteristics were determined respectively by MTT assay/colony formation assay, flow cytometry, and transmission electron microscope. In addition, the expression levels of cycle-related and apoptosis-associated proteins were individually examined by Western blot analysis. Moreover, cell proliferation and apoptosis analyses were carried out by TUNEL, Ki-67, cleaved caspase-3, and cleaved caspase-9 immunostaining assay. Our results demonstrated that IVM has a potential dosage-dependent inhibition effect on the apoptosis rate of glioma cells. Meanwhile, the results also revealed that IVM induced apoptosis by increasing caspase-3 and caspase-9 activity, upregulating the expressions of p53 and Bax, downregulating Bcl-2, activating cleaved caspase-3 and cleaved caspase-9, and blocking cell cycle in G0/G1 phase by downregulating levels of CDK2, CDK4, CDK6, cyclin D1, and cyclin E. These findings suggest that IVM has an inhibition effect on the proliferation of glioma cells by triggering cell cycle arrest and inducing cell apoptosis in vitro and in vivo, and probably represents promising agent for treating glioma.

 

https://pubmed.ncbi.nlm.nih.gov/30596403/