Anonymous ID: 474a81 Jan. 1, 2022, 5:23 p.m. No.15292927   🗄️.is đź”—kun   >>2936 >>2944 >>3034

The Leading Cause of Death for 2021 is Shocking — Covid Wasn’t Even Close

 

The leading causes of death for 2021 are now in. Covid wasn’t even close.

 

Worldometer reported thatabortion was the leading cause of death globally in 2021 with nearly 43 million killedby the procedure.

 

“As of noon on December 31, 2021, there were 42.6 million abortions performed in the course of the year, Worldometer revealed, while 8.2 million people died from cancer, 5 million from smoking, 1.7 million of HIV/AIDS, 1.3 million from traffic fatalities, and 1 million from suicide,” Breitbart reported.

 

“Totaling all the deaths in the world from causes other than abortion reveals a figure of 58.7 million, meaning that abortions accounted for just over 42 percent of all human deaths in 2021,” the report added.

 

Even if one excludes abortion, the leading causes of death in the United States show that Covid was not the highest: Cancer and heart disease were the leaders, followed by “Covid-related mortality.”

 

Here are the “average number of daily deaths” in the U.S. by leading causes of death from March 2020 to November 2021, as recorded by Statista.

 

https://beckernews.com/2-the-leading-cause-of-death-in-2021-is-shocking-covid-wasnt-even-close-43576/

Anonymous ID: 474a81 Jan. 1, 2022, 5:25 p.m. No.15292938   🗄️.is đź”—kun

==New Bombshell Study Reveals HOW Covid “Vaccines” Are Killing People

VAERS data shows the Covid "vaccines" kill people.==

 

Now, a new study makes a compelling case about how it happens.

 

We know what the problems are with the Covid vaccines, namely that they’re neither safe nor effective. Some have good theories about why they’re being pushed so hard for The Great Reset. But there have been a scant few studies that dive into how the jabs kill people. Now, we have a new study (PDF) that is shedding light on the issue.

 

In layman’s terms, the study claims the jabs confuse the immune system into attacking not just the coronavirus (which it doesn’t handle very well) but also the organs and cells of the host body itself. This is why we see athletes collapsing on the field with no preexisting health conditions shortly after they get injected. It’s why there have been millions of adverse reactions worldwide. It also explains why there have been so few autopsies performed on bodies of those who apparently died from the vaccines.

 

According to The New American:

 

A paper entitled “On COVID vaccines: why they cannot work, and irrefutable evidence of their causative role in deaths after vaccination” was published by Sucharit Bhakdi, M.D. and Arne Burkhardt, M.D., both Germany-based and widely published scientists in their fields. The findings were presented during an interdisciplinary symposium on COVID shots’ safety and efficacy on December 10, and if they received the attention and regard they deserve from health authorities, the vaccination campaign would be arguably stopped today.

 

As shown in the study, 14 of the 15 vaccinated patients who died had autoimmune damage in different organs, i.e., the patients’ immune systems were attacking their own organs.

 

The doctors noted that prior to death, only four of the 15 patients had been treated in the ICU for more than two days, while most of the patients were never hospitalized and either died at home, on the street, at work, in the car, or in home-care facilities. That fact implies that therapeutic intervention was “unlikely to have significantly influenced the post-mortem findings,” per the paper.

 

Coroners did not link the deaths to COVID vaccinations, and in most cases, “[ar]rhythmogenic heart failure” was postulated as the cause of death. Why would one’s immune system go wild and attack something it is designed to protect?

 

We’ll get to the science momentarily, but let’s piece this together with some other facts we already know and ask some serious questions. Is this why Big Pharma has been so reticent about their testing and clinical results, pushing revelations to decades out? Are doctors and scientists being silenced by the powers-that-be through bribery, bullying, and blackmail? Who knows about these effects in Washington DC? Does Anthony Fauci have anything to say about this study?

 

https://noqreport.com/2022/01/01/new-bombshell-study-reveals-how-covid-vaccines-are-killing-people/

Anonymous ID: 474a81 Jan. 1, 2022, 5:27 p.m. No.15292946   🗄️.is đź”—kun   >>2961 >>3021

Information Operation Host L Todd Wood speaks with former Overstock.com CEO Patrick Byrne on what is coming in January, 2022 in the fight against tyranny.

 

"I have faith in Durham…There's something happening in January…"

 

https://rumble.com/embed/vp5hul/?pub=4

Anonymous ID: 474a81 Jan. 1, 2022, 5:44 p.m. No.15293018   🗄️.is đź”—kun

Can Pepcid treat COVID-19?

 

With clinical trials ongoing, doctors try to unravel how famotidine could be working to fight the disease caused by SARS-CoV-2

 

Doctors and scientists are studying many existing drugs with the hope of finding therapies they can repurpose to fight COVID-19. Some of these, like Gilead Sciences’ remdesivir, directly go after the virus SARS-CoV-2, which causes the disease. Others, like Incyte’s ruxolitinib, aim to dampen the overactive immune response that characterizes later stages of disease in COVID-19.

And then there are the oddballs. Take famotidine, the active ingredient in the over-the-counter heartburn drug Pepcid. The histamine-H2-receptor antagonist works by preventing stomach acid production. That it would have any activity in an infectious disease is a bit of a head-scratcher.

Doctors first became interested in famotidine after hearing reports that people in China who took the drug for heartburn were surviving COVID-19, while other people who essentially had the same risk factors but were taking different heartburn drugs like cimetidine or omeprazole (sold in the US as Tagamet and Prilosec, respectively) were dying from the disease. Perhaps famotidine was somehow bolstering these patients and improving their chances for survival.

In early April, doctors began a clinical trial at New York’s Northwell hospitals to test that theory. They reasoned that even if evidence for famotidine’s effectiveness was largely anecdotal, the drug has been around since the 1980s and has a good safety profile. If it worked, it would be a fast and cheap way to ease the symptoms of COVID-19.

 

They decided to use high doses of intravenous famotidine. Their goal was to enroll 1,200 people with moderate to severe COVID-19 and see if those that got famotidine were less likely to die or require a ventilator. Then, in late April, the first news report about the trial appeared in Science. Boxes of Pepcid began to fly off of pharmacy shelves as people sought out any potential remedy during the pandemic.

Shortly afterward, on May 8, a team, led by Columbia University doctors Daniel Freedberg and Julian Abrams, posted a study on the preprint server medRxiv that compared the outcomes of people with COVID-19 who were prescribed famotidine within 24 hours of being admitted to the hospital to those who didn’t get the heartburn drug. They looked at the records of more than 1,600 patients at Columbia University Irving Medical Center between late February and mid-April. Of those, 84 patients received 10–40 mg of intravenous famotidine daily over the course of about 6 days.

The patients who got famotidine fared better. According to the study, they were far less likely to die or require a ventilator—a twofold decrease in risk—than those not receiving the drug. The results were published in the peer-reviewed journal Gastroenterology later in May (2020, DOI: 10.1053/j.gastro.2020.05.053).

“This is merely an association, and these findings should not be interpreted to mean that famotidine improves outcomes in patients hospitalized with COVID-19,” the team says in a statement. “It is also not clear why those patients who received famotidine had improved outcomes.”

For clarity on famotidine’s effectiveness, the team recommends awaiting the outcome of the trial going on at Northwell hospitals. “Hopefully the results from this trial will determine whether famotidine is efficacious for the treatment of COVID-19,” the team says in its statement.

Meanwhile, in early June, the journal Gut published a small case series of 10 people who developed COVID-19 and reported taking famotidine during their illness (2020, DOI: 10.1136/gutjnl-2020-321852). These people were not sick enough to go to the hospital, but their symptoms, such as cough and shortness of breath, improved within a day or two of taking the heartburn drug. It’s a small study, and the researchers acknowledge that it’s not enough to establish there’s any real benefit from taking famotidine for people who have COVID-19. Those authors recommend a clinical trial with famotidine be carried out with patients with milder disease in addition to the trial going on at Northwell hospitals.

 

https://cen.acs.org/pharmaceuticals/Pepcid-treat-COVID-19/98/i25

Anonymous ID: 474a81 Jan. 1, 2022, 5:44 p.m. No.15293022   🗄️.is đź”—kun

Pepcidantacid for muh COVID breathing problems:

 

COVID-19: Famotidine, Histamine, Mast Cells, and Mechanisms

 

Robert W. Malone, Philip Tisdall, […], and Darrell O. Ricke

 

Abstract

 

SARS-CoV-2 infection is required for COVID-19, but many signs and symptoms of COVID-19 differ from common acute viral diseases. SARS-CoV-2 infection is necessary but not sufficient for development of clinical COVID-19 disease. Currently, there are no approved pre- or post-exposure prophylactic COVID-19 medical countermeasures. Clinical data suggest that famotidine may mitigate COVID-19 disease, but both mechanism of action and rationale for dose selection remain obscure. We have investigated several plausible hypotheses for famotidine activity including antiviral and host-mediated mechanisms of action. We propose that the principal mechanism of action of famotidine for relieving COVID-19 symptoms involves on-target histamine receptor H2 activity, and that development of clinical COVID-19 involves dysfunctional mast cell activation and histamine release. Based on these findings and associated hypothesis, new COVID-19 multi-drug treatment strategies based on repurposing well-characterized drugs are being developed and clinically tested, and many of these drugs are available worldwide in inexpensive generic oral forms suitable for both outpatient and inpatient treatment of COVID-19 disease.

 

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8021898/

Anonymous ID: 474a81 Jan. 1, 2022, 6:03 p.m. No.15293112   🗄️.is đź”—kun

>>15293096

I quit summer 2009. It was one of the best decisions I’ve ever made. It’s hard at first, especially when I’m social situations where it seems everyone else is drinking, but you have the will within you. It is definitely beneficial to your health to quit. By the Grace of my Lord and Savior Jesus Christ I am approaching 13 years sober!

Anonymous ID: 474a81 Jan. 1, 2022, 6:17 p.m. No.15293191   🗄️.is đź”—kun

>>15293155

I’m convinced that Omicron is the death knell for COVID. It will provide the herd immunity that TPTB claimed they were seeking but, in reality, we’re counter acting all along with their mandates, vaccines, and lockdowns of healthy people. Once the masses of sleepy sheep get Omicron, and most recover, and see for themselves that COVID isn’t the monster the media, healthcare, and government made it out to be, COVID will lose its boogie man status. TPTB will likely try to keep the hysteria going but, if enough people experience this for themselves, I think the gig wil be up.