Anonymous ID: 396549 Jan. 5, 2022, 8:21 p.m. No.15317729   🗄️.is 🔗kun

>>15317707

Who disfigured or tortured children.

I made a joke.

The joke was poking fun at the state of this board (depending on who’s bakering).

Chill the fuck out or I will find you, and meme the fuck out of you.

Anonymous ID: 396549 Jan. 5, 2022, 8:46 p.m. No.15317845   🗄️.is 🔗kun

>>15317823

Difficult comparison.

PET scan lights up (dark areas) where high metabolic activity is. The tracer is radioactive glucose. Stupid time to scan 8 days after booster vaccination given.

Anonymous ID: 396549 Jan. 5, 2022, 8:53 p.m. No.15317885   🗄️.is 🔗kun

>>15317859

My criticism remains.

Activating immunologic cells with a vaccine booster is not showing cancer cells unless this is some scan modality NOT using radioactive glucose as the tracer molecule.

Anonymous ID: 396549 Jan. 5, 2022, 8:59 p.m. No.15317910   🗄️.is 🔗kun

>>15317859

This here pasta from the article supports the connection your post expressed concern over. Namely that the mRNA vaccine can result in unintended expansion of an uncommon T cell lymphoma.

 

A 18F-FDG PET/CT revealed multiple voluminous hypermetabolic lymphadenopathies above and below the diaphragm as well as several extra-nodal hypermetabolic lesions (Figure 1, left panel). Considering a presumptive diagnosis of stage IV lymphoma, a left cervical lymph node biopsy was performed. Pathological examination revealed residual atrophic germinal centers, surrounded by an expanded paracortical area composed of an atypical T-cell infiltrate with clear cell morphology, expressing TFH cell markers (CD3, CD4, PD1, ICOS, BCL6, CXCL13) and a loss of CD7. The paracortical area contained an increased number of high-endothelial venules, supported by an increased number of follicular dendritic cell networks, with some foci of EBV+ B-cell immunoblastic proliferation in the background (Figure 2). These features highly suggested a diagnosis of AngioImmunoblastic T cell Lymphoma (AITL), pattern 2. Next generation sequencing (NGS) performed on the biopsy specimen identified the RHOA G17V mutation characteristic of AITL (5) together with the DNMT3A, IDH2 and TET2 mutations. A TCR-gamma gene rearrangement confirmed a clonal T cell proliferation. Altogether, these findings unambiguously established the diagnosis of AITL. A bone marrow biopsy did not reveal neither morphological nor phenotypic abnormalities, but NGS revealed DMNT3A and TET2 mutations in bone marrow cells with allele frequencies of 41% and 36%, respectively.