Anonymous ID: 200f7d May 14, 2022, 5:12 a.m. No.16272540   🗄️.is 🔗kun   >>2562 >>2696 >>2944

https://twitter.com/KimDotcom/status/1525293982133407744

 

https://threadreaderapp.com/thread/1525293982133407744.html

 

Kim Dotcom

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9h • 13 tweets • 3 min read

🧵 This may be the most important threat that I ever make. Big picture stuff.

 

I’ll try to help you understand why the future is not what we’re hoping for. A major global collapse is coming. It may be worse than we can imagine.

 

Our leaders know.

But what are they planning?

The United States did not have a surplus or a balanced budget since 2001 and in the last 50 years it only had 4 years of profit. In fact all profit the US had in the last 50 years wouldn’t be enough to pay for 6 months of the current yearly deficit. So what do they do?

Image

US spending and debt have spiraled out of control and the Govt can only raise the money it needs by printing it.

 

That causes inflation. It’s like taxing you extra because you pay more for the things you need and all your assets decline in value.

 

See the money printing frenzy:

Image

The problem is that this has been going on for so long there’s now no way to fix it.

 

The reality is that the US has been bankrupt for some time and what’s coming is a nightmare: Mass poverty and a new system of control.

 

Let me explain why this isn’t just doom and gloom talk.

Total US debt is at $90 trillion. US unfunded liabilities are at $169 trillion. Combined that’s $778,000 per US citizen or $2,067,000 per US tax payer.

 

Remember, the only way the Government can operate now is by printing more money. Which means hyperinflation is inevitable.

The total value of ALL companies listed on the US stock market is $53 trillion.

 

The real value is much lower because the US has been printing trillions to provide interest free loans to investment banks to pump up the stock market. It’s a scam.

 

Most of the $53 trillion is air.

The value of all US assets combined, every piece of land, real estate, all savings, all companies, everything that all citizens, businesses, entities and the state own is worth $193 trillion.

 

That number is also full of air just like the US stock market.

Let’s do the math:

 

US total debt

$90 trillion

 

US unfunded liabilities

$169 trillion

 

Total

$259 trillion

 

Minus all US assets

$193 trillion

 

Balance

  • $66 trillion

 

That’s $66 trillion of debt and liabilities after every asset in the US has been sold off.

 

Do you understand?

So even if the US could sell all its assets at the current value, which is impossible, it would still be broke.

 

The US is beyond bankrupt.

This patient is already dead.

This patient is now a zombie.

 

You probably wonder why are things still going? Why didn’t it all collapse yet.

It’s all perception and denial.

 

The perception is that the US has the largest economy and the strongest military in the world. But in reality the US is broke and can’t afford its army.

 

The denial is that all nations depend on a strong US or else the global markets will crash.

The only reason why the US zombie keeps going is because the end of the US is the end of western prosperity and an admission that capitalism failed as a model for the world.

 

But it doesn’t change the reality. The collapse is inevitable and coming.

 

What’s the plan to fix this?

You may have heard of the great reset or the world government.

 

Is it a controlled demolition of the global markets, economies and the world as we know it?

 

A shift into a new dystopian future where the elites are the masters of the slaves without the cosmetics of democracy?

Without a controlled demolition the world will collapse for all, including the elites.

 

The world has changed so much, nothing seems to make sense anymore, the blatant corruption is out in the open, the obvious propaganda media, the erosion of our rights.

 

What’s the end game?

Anonymous ID: 200f7d May 14, 2022, 5:42 a.m. No.16272649   🗄️.is 🔗kun   >>2651

re: formula AND kidney issue?

 

"Melamine is a chemical compound with a wide array of uses, including fertilizers, resins, construction materials, and flame retardants.

 

China is the main producer of this synthetic compound, and melamine-containing products are exported around the world.

 

Melamine is frequently found in drinking water. It has been detected in breast milk and in children’s urine, both in Asia and in the US.

 

Melamine may be best known as the center of a 2008 scandal in China, where dozens of food companies were found to have added relatively pure melamine to milk powder to increase the protein content. This milk powder was used in infant formula, and thousands of babies became ill as a result.

 

Melamine has also been added to pet food products to artificially increase the protein reading. This resulted in thousands of deaths of cats and dogs in 2007, and one of the largest-ever pet food recalls in the US."

 

It’s Time To Think About Alternatives To Melamine (Which May Be In The Plate You’re Using Right Now)

https://www.forbes.com/sites/christinero/2019/11/29/its-time-to-think-about-alternatives-to-melamine-which-may-be-in-the-plate-youre-using-right-now/?sh=564590b73964

 

“Of an estimated 300,000 victims in China, six babies died from kidney stones and other kidney damage and an estimated 54,000 babies were hospitalized.“

 

The 2008 Chinese milk scandal

https://en.m.wikipedia.org/wiki/2008_Chinese_milk_scandal

 

“Beginning in March 2007, there was a wide recall of many brands of cat and dog foods due to contamination with melamine and cyanuric acid. The recalls in North America, Europe, and South Africa came in response to reports of kidney failure in pets.

 

…many sources speculate the actual number of affected pets may never be known, and experts are concerned that the actual death toll could potentially reach into the thousands.”

 

2007 pet food recalls

https://en.m.wikipedia.org/wiki/2007_pet_food_recalls

 

You can't imagine how many medication recalls there are because of cancer causing ingredients in the meds.

I'm not sure if they all come from China though.

 

The coronavirus outbreak exposes the U.S.’s pharma supply chain vulnerability

https://www.statnews.com/2020/02/14/coronavirus-outbreak-exposes-weak-link-us-drug-supply-chain/

Anonymous ID: 200f7d May 14, 2022, 5:43 a.m. No.16272651   🗄️.is 🔗kun   >>2653 >>2679

>>16272649

July 27, 2021

 

A Breakthrough in Infant Formula: 2'-FL HMO

 

Pioneering Abbott research helps bring our baby formula closer than ever to breast milk.

 

2'-FL HMO, or 2'-fucosyllactose (few-co-syl-lactose) human milk oligosaccharide (ol-i-goh-sak-uh-rahyd); is a prebiotic found naturally in breast milk.

 

About 80 percent of mothers make 2'-FL in their breast milk. Excluding water, HMOs are the third most abundant ingredient in breast milk after fat and carbohydrates.

 

What is an HMO's job?

 

Simply, they help the good bacteria do the good things you want in your baby's gut. 2'-FL HMO also travels through the bloodstream.

 

"We wanted to replicate HMOs so that babies who drink formula can benefit from them," Rachael Buck, Ph.D., associate research fellow and immunology expert at Abbott, said.

 

Similac Pro-Advance, Similac Pro-Sensitive and Similac Pro-Total Comfort all have 2'-FL HMO. The 2'-FL HMO in Abbott's formulas is bio-structurally identical to the 2’-FL HMO found in mother's milk and it is produced in a similar way to the way some vitamins are made.

https://www.abbott.com/corpnewsroom/products-and-innovation/HMO-fundamentals.html

Anonymous ID: 200f7d May 14, 2022, 5:43 a.m. No.16272653   🗄️.is 🔗kun   >>2679

>>16272651

July 27, 2021

 

A Breakthrough in Infant Formula: 2'-FL HMO

 

Pioneering Abbott research

helps bring our baby formula

closer than ever to breast milk.

 

2'-FL HMO,

or 2'-fucosyllactose

(few-co-syl-lactose)

human milk oligosaccharide

(ol-i-goh-sak-uh-rahyd);

is a prebiotic

found naturally in breast milk.

 

About 80 percent of mothers

make 2'-FL in their breast milk.

 

Excluding water,

HMOs are the third most abundant ingredient

in breast milk

after fat and carbohydrates.

 

What is an HMO's job?

 

Simply, they help the good bacteria

do the good things you want

in your baby's gut.

 

2'-FL HMO also travels through the bloodstream.

 

"We wanted to replicate HMOs

so that babies who drink formula

can benefit from them,"

Rachael Buck, Ph.D.,

associate research fellow

and immunology expert at Abbott, said.

 

Similac Pro-Advance,

Similac Pro-Sensitive

and Similac Pro-Total Comfort

all have 2'-FL HMO.

 

The 2'-FL HMO in Abbott's formulas

is bio-structurally identical

to the 2’-FL HMO found in mother's milk

and it is produced in a similar way

to the way some vitamins are made.

Anonymous ID: 200f7d May 14, 2022, 5:48 a.m. No.16272679   🗄️.is 🔗kun

>>16272651

>>16272653

 

Human Milk Components Modulate Toll-Like Receptor–Mediated Inflammation

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4717889/

 

Toll-like receptor (TLR) signaling is central to innate immunity. Aberrant expression of TLRs is found in neonatal inflammatory diseases. Several bioactive components of human milk modulate TLR expression and signaling pathways, including soluble toll-like receptors (sTLRs), soluble cluster of differentiation (sCD) 14, glycoproteins, small peptides, and oligosaccharides. Some milk components, such as sialyl (α2,3) lactose and lacto-N-fucopentaose III, are reported to increase TLR signaling; under some circumstances this might contribute toward immunologic balance. Human milk on the whole is strongly anti-inflammatory, and contains abundant components that depress TLR signaling pathways: sTLR2 and sCD14 inhibit TLR2 signaling; sCD14, lactadherin, lactoferrin, and 2′-fucosyllactose attenuate TLR4 signaling; 3′-galactosyllactose inhibits TLR3 signaling, and β-defensin 2 inhibits TLR7 signaling. Feeding human milk to neonates decreases their risk of sepsis and necrotizing enterocolitis. Thus, the TLR regulatory components found in human milk hold promise as benign oral prophylactic and therapeutic treatments for the many gastrointestinal inflammatory disorders mediated by abnormal TLR signaling.

 

Keywords: immune function, toll like receptors, inflammation, human milk, glycans

 

Human milk is a putative innate immune system

 

Conclusions

TLR signaling is central to innate immunity, and neonatal inflammatory diseases involve the loss of TLR signaling homeostatic control. Human milk contains components, including sTLRs, sCD14, glycoproteins, small peptides, and oligosaccharides, that modulate the immune system and suppress inflammation. In contradistinction, 3SL, LNFP III, and a glycoprotein of >80 kDa increase TLR signaling, but these could contribute toward balancing complex signaling networks of the innate immune system. Beyond initiating inflammatory processes through NF-κB cascades, TLRs also function in mucosal homeostasis and inflammation through the activation of innate and adaptive immunity, promotion of cell proliferation, maintenance of the mucosal intestinal epithelial barrier, and coordination of mucosal homeostasis. Human milk on the whole is strongly anti-inflammatory. sTLR2 and sCD14 inhibit TLR2 signaling; sCD14, lactadherin, lactoferrin, and 2′-fucosyllactose inhibit TLR4 signaling; 3′-galactosyllactose inhibits TLR3 signaling, and β-defensin 2 inhibits TLR7 signaling, thereby quenching inflammation at the mucosal surface. Feeding human milk to neonates decreases their risk of sepsis and NEC. TLR regulatory components in human milk hold promise as benign oral prophylactic and therapeutic treatments for the many gastrointestinal inflammatory disorders mediated by abnormal TLR signaling. Their molecular characterization and synthesis will allow preclinical and clinical studies to test their efficacy for the prevention or amelioration of NEC, neonatal sepsis, inflammatory bowel diseases, and other gastrointestinal inflammatory disorders of diverse etiologies.

Anonymous ID: 200f7d May 14, 2022, 5:50 a.m. No.16272690   🗄️.is 🔗kun

Toll-like receptor-induced inflammatory cytokines

are suppressed by Gain of function

or overexpression of Gai2 protein

 

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3563060/

 

Abstract

Previous studies have implicated a role of Gαi proteins as co-regulators of Toll –like receptor (TLR) activation. These studies largely derived from examining the effect of Gαi protein inhibitors or genetic deletion of Gαi proteins. However the effect of increased Gαi protein function or Gαi protein expression on TLR activation has not been investigated. We hypothesized that gain of function or increased expression of Gαi proteins suppresses TLR2 and TLR4 -induced inflammatory cytokines. Novel transgenic mice with genomic “knock-in” of a Regulator of G-protein Signaling (RGS)-insensitive Gnai2 allele (Gαi2 G184S/G184S; GS/GS) were employed. These mice express essentially normal levels of Gαi2 protein, however the Gαi2 is insensitive to its negative regulator RGS thus rendering more sustained Gαi2 protein activation following ligand/receptor binding. In subsequent studies, we generated Raw 264.7 cells that stably overexpress Gαi2 protein (Raw Gαi2). Peritoneal macrophages, splenocytes and mouse embryonic fibroblasts (MEF) were isolated from WT and GS/GS mice and were stimulated with LPS, Pam3CSK4 or Poly (I:C). We also subjected WT and GS/GS mice to endotoxic shock (LPS 25mg/kg i.p.) and plasma TNFα and IL-6 production were determined. We found that in vitro LPS and Pam3CSK4 induced TNFα and IL-6 production are decreased in macrophages from GS/GS mice compared with WT mice (p<0.05). In vitro LPS and Pam3CSK4 induced IL-6 production in splenocytes and in vivo LPS induced IL-6 were suppressed in GS/GS mice. Poly (I:C) induced TNFα and IL-6 in vitro demonstrated no difference between GS/GS mice and WT mice. LPS induced IL-6 production was inhibited in MEFs from GS/GS mice similarly to macrophage and splenocytes. In parallel studies, Raw Gαi2 cells also exhibit decreased TNFα and IL-6 production in response to LPS and Pam3CSK4. These studies support our hypothesis that Gαi2 proteins are novel negative regulators of TLR activation.