Experts Puzzled By Why Haiti Has One of the Lowest COVID-19 Death Rates In the World Despite Administering Zero Vaccine Doses: ‘We Don’t Know’

https://atlantablackstar.com/2021/05/11/experts-puzzled-by-why-haiti-has-one-of-the-lowest-covid-19-death-rates-in-the-world-despite-administering-zero-vaccine-doses-we-dont-know/

For months, health experts have tried to pinpoint the reason for Haiti’s inexplicitly low COVID-19 death rate as other countries continue to struggle to contain exploding numbers of cases and deaths. In India, a global hotspot for the virus, the seven-day average for cases has reached 385,000, while deaths have routinely topped 3,000 a day in the nation of nearly 1.4 billion people.

In Haiti, a country with a population of more than 11 million, there have been 13,164 cases throughout the entire pandemic, and 264 deaths. The Caribbean nation has one of the lowest COVID death rates in the world, at just 22 per million. In Delhi, that number reached 892 in April before the outbreak continued to accelerate. In the United States, that figure stands at 1,800 per million, and at nearly 3,000 per million in some places in Europe.

Children in Haiti. Stock photo (Credit: Pexels)

Dr. Jean “Bill” Pape, a top Haitian infectious disease expert, said the nation’s low death rate can’t be attributed to strict adherence to mask-wearing or social distancing guidelines.

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“Most people don’t wear a mask,” he said.

By December, most restrictions had been lifted and buses and markets have been crowded in recent months. No one in Haiti has received a single dose of a coronavirus vaccine.

When COVAX, a program run by the World Health Organization, offered Haiti AstraZeneca vaccines, the government declined the offer.

“Because COVID did not impact us as badly, people don’t think it [the vaccine] is worth it actually,” Dr. Jacqueline Gautier, a member of Haiti’s advisory group on COVID vaccinations, told NPR.

According to Pape, there has not been a resurgence of cases. “Sometimes it’s two, sometimes zero, sometimes it’s 20 cases,” he said. “But we are not seeing a second wave, as we had thought would happen.”

Last summer cases rose sharply in Haiti but still never reached critical levels, Ministry of Health data shows. By the fall, the health agency Pape heads closed its COVID units because there were too few patients. Other units have closed in recent months and plans to turn soccer stadiums into oversized hospitals were never executed.

Dr. Sophia Cherestal Wooley, deputy medical coordinator for Doctors Without Borders/Médecins Sans Frontières in Port-au-Prince, spoke with the Tampa Bay Times about Haiti’s low number of cases and death in December.

“We don’t have a large quantity of people who are in bad shape,” she said. “They don’t get sick to the point that they need to be hospitalized and we don’t have the same quantity of people who have died here like in the Dominican Republic.” (Haiti shares the island of Hispaniola with the Dominican Republic.) She added, “Sincerely, I don’t have an explanation as to why,”

Speaking to NPR, Gautier said Haiti’s young population has protected it from higher numbers of coronavirus cases and deaths. The average of the population is just 23, and younger [people are less likely to suffer from severe symptoms of the virus. In addition, homes in Haiti are constructed in an open fashion that allows for significant ventilation, which has been shown to reduce indoor transmission of COVID.

While the precise reasons for the low number of cases and deaths in Haiti remain hard to identify, The Pan American Health Organization announced plans last year to test Haitians for COVID antibodies to determine what percentage of the population has already contracted the virus.

Chief Nerd

@chiefnerd

·

Dec 25

The aftermath of the protests in Paris yesterday. CBC says they were “largely peaceful”.

https://gettr.com/post/p22wrxbe78a

>>18023035

>>18023043

Race. Bloodline. Unique haplogroup

1250 COVID Vaccine Publications and Case Reports

Collection of peer reviewed case reports and studies citing adverse effects post COVID vaccination.

Researching Covid vaccine adverse events can be daunting in part due to a broad myriad of factors. Primarily, the information is incredibly challenging to find. Here, we share an ever growing list of peer-reviewed studies specific to Covid vaccine adverse events. This list is curated and maintained by our dedicated staff of injured PhDs and medical professionals.

Before diving in, please take a look at our Research Primer: How to Read and Understand Research for tools to how best approach the massive amount of information found in the document below. As always, this is for informational purposes only. Please discuss with your trusted medical team.

Time to assume that health research is fraudulent until proven otherwise?

https://blogs.bmj.com/bmj/2021/07/05/time-to-assume-that-health-research-is-fraudulent-until-proved-otherwise/

Health research is based on trust. Health professionals and journal editors reading the results of a clinical trial assume that the trial happened and that the results were honestly reported. But about 20% of the time, said Ben Mol, professor of obstetrics and gynaecology at Monash Health, they would be wrong. As I’ve been concerned about research fraud for 40 years, I wasn’t that surprised as many would be by this figure, but it led me to think that the time may have come to stop assuming that research actually happened and is honestly reported, and assume that the research is fraudulent until there is some evidence to support it having happened and been honestly reported. The Cochrane Collaboration, which purveys “trusted information,” has now taken a step in that direction.

As he described in a webinar last week, Ian Roberts, professor of epidemiology at the London School of Hygiene & Tropical Medicine, began to have doubts about the honest reporting of trials after a colleague asked if he knew that his systematic review showing the mannitol halved death from head injury was based on trials that had never happened. He didn’t, but he set about investigating the trials and confirmed that they hadn’t ever happened. They all had a lead author who purported to come from an institution that didn’t exist and who killed himself a few years later. The trials were all published in prestigious neurosurgery journals and had multiple co-authors. None of the co-authors had contributed patients to the trials, and some didn’t know that they were co-authors until after the trials were published. When Roberts contacted one of the journals the editor responded that “I wouldn’t trust the data.” Why, Roberts wondered, did he publish the trial? None of the trials have been retracted.

Later Roberts, who headed one of the Cochrane groups, did a systematic review of colloids versus crystalloids only to discover again that many of the trials that were included in the review could not be trusted. He is now sceptical about all systematic reviews, particularly those that are mostly reviews of multiple small trials. He compared the original idea of systematic reviews as searching for diamonds, knowledge that was available if brought together in systematic reviews; now he thinks of systematic reviewing as searching through rubbish. He proposed that small, single centre trials should be discarded, not combined in systematic reviews.

Mol, like Roberts, has conducted systematic reviews only to realise that most of the trials included either were zombie trials that were fatally flawed or were untrustworthy. What, he asked, is the scale of the problem? Although retractions are increasing, only about 0.04% of biomedical studies have been retracted, suggesting the problem is small. But the anaesthetist John Carlisle analysed 526 trials submitted to Anaesthesia and found that 73 (14%) had false data, and 43 (8%) he categorised as zombie. When he was able to examine individual patient data in 153 studies, 67 (44%) had untrustworthy data and 40 (26%) were zombie trials. Many of the trials came from the same countries (Egypt, China, India, Iran, Japan, South Korea, and Turkey), and when John Ioannidis, a professor at Stanford University, examined individual patient data from trials submitted from those countries to Anaesthesia during a year he found that many were false: 100% (7/7) in Egypt; 75% (3/ 4) in Iran; 54% (7/13) in India; 46% (22/48) in China; 40% (2/5) in Turkey; 25% (5/20) in South Korea; and 18% (2/11) in Japan. Most of the trials were zombies. Ioannidis concluded that there are hundreds of thousands of zombie trials published from those countries alone.

p1

>>18023065

Others have found similar results, and Mol’s best guess is that about 20% of trials are false. Very few of these papers are retracted.

We have long known that peer review is ineffective at detecting fraud, especially if the reviewers start, as most have until now, by assuming that the research is honestly reported. I remember being part of a panel in the 1990s investigating one of Britain’s most outrageous cases of fraud, when the statistical reviewer of the study told us that he had found multiple problems with the study and only hoped that it was better done than it was reported. We asked if had ever considered that the study might be fraudulent, and he told us that he hadn’t.

We have now reached a point where those doing systematic reviews must start by assuming that a study is fraudulent until they can have some evidence to the contrary. Some supporting evidence comes from the trial having been registered and having ethics committee approval. Andrew Grey, an associate professor of medicine at the University of Auckland, and others have developed a checklist with around 40 items that can be used as a screening tool for fraud (you can view the checklist here). The REAPPRAISED checklist (Research governance, Ethics, Authorship, Plagiarism, Research conduct, Analyses and methods, Image manipulation, Statistics, Errors, Data manipulation and reporting) covers issues like “ethical oversight and funding, research productivity and investigator workload, validity of randomisation, plausibility of results and duplicate data reporting.” The checklist has been used to detect studies that have subsequently been retracted but hasn’t been through the full evaluation that you would expect for a clinical screening tool. (But I must congratulate the authors on a clever acronym: some say that dreaming up the acronym for a study is the most difficult part of the whole process.)

Roberts and others wrote about the problem of the many untrustworthy and zombie trials in The BMJ six years ago with the provocative title: “The knowledge system underpinning healthcare is not fit for purpose and must change.” They wanted the Cochrane Collaboration and anybody conducting systematic reviews to take very seriously the problem of fraud. It was perhaps coincidence, but a few weeks before the webinar the Cochrane Collaboration produced guidelines on reviewing studies where there has been a retraction, an expression of concern, or the reviewers are worried about the trustworthiness of the data.

Retractions are the easiest to deal with, but they are, as Mol said, only a tiny fraction of untrustworthy or zombie studies. An editorial in the Cochrane Library accompanying the new guidelines recognises that there is no agreement on what constitutes an untrustworthy study, screening tools are not reliable, and “Misclassification could also lead to reputational damage to authors, legal consequences, and ethical issues associated with participants having taken part in research, only for it to be discounted.” The Collaboration is being cautious but does stand to lose credibility—and income—if the world ceases to trust Cochrane Reviews because they are thought to be based on untrustworthy trials.

p2

>>18023069

Research fraud is often viewed as a problem of “bad apples,” but Barbara K Redman, who spoke at the webinar insists that it is not a problem of bad apples but bad barrels if not, she said, of rotten forests or orchards. In her book Research Misconduct Policy in Biomedicine: Beyond the Bad-Apple Approach she argues that research misconduct is a systems problem—the system provides incentives to publish fraudulent research and does not have adequate regulatory processes. Researchers progress by publishing research, and because the publication system is built on trust and peer review is not designed to detect fraud it is easy to publish fraudulent research. The business model of journals and publishers depends on publishing, preferably lots of studies as cheaply as possible. They have little incentive to check for fraud and a positive disincentive to experience reputational damage—and possibly legal risk—from retracting studies. Funders, universities, and other research institutions similarly have incentives to fund and publish studies and disincentives to make a fuss about fraudulent research they may have funded or had undertaken in their institution—perhaps by one of their star researchers. Regulators often lack the legal standing and the resources to respond to what is clearly extensive fraud, recognising that proving a study to be fraudulent (as opposed to suspecting it of being fraudulent) is a skilled, complex, and time consuming process. Another problem is that research is increasingly international with participants from many institutions in many countries: who then takes on the unenviable task of investigating fraud? Science really needs global governance.

Everybody gains from the publication game, concluded Roberts, apart from the patients who suffer from being given treatments based on fraudulent data.

Stephen Lock, my predecessor as editor of The BMJ, became worried about research fraud in the 1980s, but people thought his concerns eccentric. Research authorities insisted that fraud was rare, didn’t matter because science was self-correcting, and that no patients had suffered because of scientific fraud. All those reasons for not taking research fraud seriously have proved to be false, and, 40 years on from Lock’s concerns, we are realising that the problem is huge, the system encourages fraud, and we have no adequate way to respond. It may be time to move from assuming that research has been honestly conducted and reported to assuming it to be untrustworthy until there is some evidence to the contrary.

Richard Smith was the editor of The BMJ until 2004.

Competing interest: RS was a cofounder of the Committee on Medical Ethics (COPE), for many years the chair of the Cochrane Library Oversight Committee, and a member of the board of the UK Research Integrity Office.

3 of 3

Mainland Chinese vaccine tourists flock to Macau for mRNA Covid jabs

https://www.ft.com/content/d89551a9-4f7e-434f-94d8-05c3dc93da81

(paywall)

Tromethamine is a Hazardous Substance in Jabs that Must be Banned

Pfizer added Tromethamine to it jabs without any clinical trials as a replacement for Phosphate buffer used in it original 4 formulations.

https://geoffpain.substack.com/p/tromethamine-is-a-hazardous-substance

Tromethamine is an Endocrine Disruptor and causes Anaphylaxis.

In Australia the Therapeutic Goods Administration, acting on the advice of The Advisory Committee on Vaccines, and The Australian Technical Advisory Group on Immunisation, approved a modified Pfizer injection for young Children aged 5-11 years old, using Tromethamine, also known as Tris or THAM as an ingredient.

Its Chemical name tris(hydroxymethyl)aminomethane and it is used as pH buffer.

The approval was given for use of the Pfizer vaccine despite the fact that no trials were conducted with the modified product. The only trial in this age group was done with Phosphate buffering.

Australian advisors know it can cause life threatening conditions many of which appear in the Database of Adverse Event Notifications.

Deaths have been reported following administration of this vaccine, including a 7 year old boy who died of Tonic-Clonic Seizure and Cardiac Arrest.

Deaths associated with use of other products in Australia incorporating Tromethamine are known.

50 years ago it was known that Tromethamine affects your Myocardial Heart muscle by reducing its contractility. Study used isolated Cat papillary muscles. Still behind a paywall.

https://journals.physiology.org/doi/abs/10.115/ajplegacy.1972.222.1.10?journalCode=ajplegacy

60 years ago it was known that Tromethamine interferes with Kidney function.

Tromethamine is converted by your red blood cells to the toxic Phosphate derivative [2-amino-3-hydroxy-2-(hydroxymethyl)propyl] dihydrogen phosphate, aka Phosphoryl-Tris. Published in 1970, experts in vaccines apparently are unaware of this hazard. Known enzyme inhibitor.

Tromethamine is a flexible toxin that can fit into numerous DNA and enzyme sites in your body, as shown in numerous crystal structures of its acid salts with your organic acids.

p1

>>18023084

https://www.sciencedirect.com/science/article/abs/pii/S002228602030137X

In 1972 it was known that Tromethamine inhibits numerous enzymes including Threonine Dehydratase

https://www.researchgate.net/publication/18106518_Inhibition_of_Rat_Liver_Threonine_Dehydratase_by_Trishydroxymethyl_aminomethane_Tris_and_Reversal_by_Orthophosphate

In 1977 it was demonstrated that Tromethamine is an Endocrine Disruptor, upsetting your Thyroid Hormones. "The Golgi zone is that region in the parathyroid gland in which proparathyroid hormone to parathyroid hormone conversion is initiated and that Tris inhibits this conversion through disruption of the converting site."

https://www.researchgate.net/publication/22285955_Disruption_of_the_Golgi_zone_and_inhibition_of_the_conversion_of_proparathyroid_hormone_to_parathyroid_hormone_in_human_parathyroid_tissue_by_Trishydroxymethylaminomethane

Tromethamine inhibits not only the sialic acid addition which takes place in the trans Golgi region, but also the trimming step of high mannose-type oligosaccharides

https://www.researchgate.net/publication/20111404_Tris_inhibits_both_proteolytic_and_oligosaccharide_processing_occuring_in_the_Golgi_complex_in_primary_culture_rat_hepatocytes

In this Phospholipase A2 enzyme (PLA2), 4 Tris molecules were found lurking by x-ray crystal structure determination.

Three were binding to and altering the shape via Hydrogen bonding with the protein chain.

The 4th Tris molecule was found coordinating to one of the 3 Calcium ions. PLA2s are found in numerous organs, including your Pancreas, Spleen, Thymus, Lung, Colon, Brain, Macrophages, Platelets and Monocytes. They play important roles in your Inflammatory Response and Blood Platelet Aggregation.

Jabbing Tromethamine into your body will have consequences.

p2

>>18023085

https://www.researchgate.net/publication/7227183_Third_calcium_ion_found_in_an_inhibitor-bound_phospholipase_A2

In 1986 it was known that Tromethamine inhibits Dextransucrase.

https://www.researchgate.net/publication/20213480_Inhibition_of_dextransucrase_by_Zn2_Ni2_Co2_and_Trishydroxymethylaminomethane_Tris

Tromethamine is known to block ion channels which points to its possible role in developing Amyloid Disease.

https://www.researchgate.net/publication/14817949_Alzheimer_Disease_Amyloid_beta_Protein_Forms_Calcium_Channels_in_Bilayer_Membranes_Blockade_by_Tromethamine_and_Aluminum

The US Comparative Toxicogenomics Database (CTD) has a page for Tromethamine showing that the top 2 of your genes that it interferes with are the Amyloid Precursor Protein (APP) gene, essential in Brain development before and after birth, and the Albumin (ALB) gene that codes for plasma transporter proteins that carry most of your Zinc, Calcium and Magnesium. Other functions of these genes are still being worked out.

http://ctdbase.org/detail.go?type=chem&acc=D014325

Tromethamine inhibits Phospholipase C enzymes, which are important in signal transduction.

https://www.researchgate.net/publication/20661728_High-resolution_15_A_crystal_structure_of_phospholipase_C_from_Bacillus_cereus

https://www.researchgate.net/publication/14684775_The_Crystal_Structure_of_Tris-inhibited_Phospholipase_C_from_Bacillus_cereus_at_19_A_Resolution

It is known with atomic precision exactly how Tromethamine inhibits Zinc enzymes, as shown 20 years ago in the x-ray crystal structure of Aminopeptidase from Aeromonas proteolytica. "Bimetallohydrolases catalyze such diverse reactions as the degradation of DNA, RNA, phospholipids, and polypeptides making them key players in carcinogenesis, tissue repair, protein maturation, cell cycle control, Hormone-level regulation, and protein degradation. These enzymes are also involved in the degradation of agricultural Neurotoxins, Urea, Antibiotics, and several phosphorous(V) materials used in Chemical Weaponry".

https://www.cell.com/structure/fulltext/S0969-2126(02)00810-9

Tromethamine can permanently deactivate Alanine Racemase enzymes.

Deactivation of Alanine Racemase by Tromethamine is catalytic, i.e. one molecule can attack and destroy multiple enzyme molecules.

The authors propose this scheme as the mechanism, see Tris entering at the top left and emerging bottom right.

Humans don't have this particular Racemase, so it will be interesting to find out which Human Racemase enzymes are affected by this jab component.

p3

>>18023086

https://www.researchgate.net/publication/332288348_Cold-induced_aldimine_bond_cleavage_by_Tris_in_Bacillus_subtilis_alanine_racemase

Tromethamine binds with Lysozyme.

https://www.sciencedirect.com/science/article/abs/pii/S0003269708002121

Tromethamine inhibits the processing and secretion of albumin and α-1 antitrypsin by hepatocytes in culture.

https://febs.onlinelibrary.wiley.com/doi/abs/10.1016/0014-5793(90)81415-K

Tromethamine (Tris) inhibits not only Insulin secretion but also the conversion from Proinsulin to Insulin, as demonstrated 40 years ago.

https://febs.onlinelibrary.wiley.com/doi/abs/10.1016/0014-5793(90)81415-K

Tromethamine suppresses Glucose-Induced Insulin release.

https://www.diabetesresearchclinicalpractice.com/article/S0168-8227(85)80031-0/pdf

In 1970 it was suggested that the known ability of Tromethamine to complex Zinc ions might assist it to influence release of stored Insulin.

https://www.researchgate.net/publication/18254150_Tris_Hydroxymethyl_Aminomethane_THAM_Induced_Stimulation_of_Insulin_Release_by_Islets_of_Langerhans_Previously_Isolated_from_Rat_Pancreas

In 1986, use of radioactive 3H-Leucine enabled the more detailed study of the impact of Tromethamine on Insulin release and Pancreatic Protein synthesis.

https://diabetesjournals.org/diabetes/article/35/4/433/7807/Tris-hydroxymethyl-aminomethane-Inhibits-the

Tromethamine prevents effective treatment of Escherichia coli with the Myeloperoxidase-Hydrogen Peroxide-Chloride Antimicrobial System because it forms high-molecular-weight, charged, or polar N-Cl derivatives that would be unable to penetrate the cell membrane.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC414428/

Tromethamine easily forms numerous new chemicals with undetermined toxicity. In this example, 2-aldehyde-8-hydroxyquinoline forms condensation compounds in the presence of Lanthanide elements.

https://www.sciencedirect.com/science/article/abs/pii/S0022286014010461

Tromethamine increase NETosis, an important factor in Blood Clots.

https://www.frontiersin.org/articles/10.3389/fmed.2018.00019/full

Tromethamine inactivates the bacterial enzyme 5'-MethylThioadenosine/S-Adenosylhomocysteine Nucleosidase (MTAN) which plays a central role in three essential metabolic pathways in bacteria: methionine salvage, purine salvage, and polyamine biosynthesis. Giving more clues about what it might do in the Human body.

https://www.researchgate.net/publication/47451464_Enzyme-ligand_interactions_that_drive_active_site_rearrangements_in_the_Helicobacter_pylori_5'-methylthioadenosineS-adenosylhomocysteine_nucleosidase

Interesting electrochemical degradation products of Tromethamine were identified in high voltage electrophoresis of ssDNA with the authors reverting to Phosphate Buffer to prevent the problem.

https://pubmed.ncbi.nlm.nih.gov/28251659/

Tromethamine forms stable adducts with a number of Chemical Warfare Agents, including the Fluoride poison SARIN.

https://pubmed.ncbi.nlm.nih.gov/20172768/

Tromethamine (TRIS) has been shown to interfere with Human Vitamin B6 (Pyridoxal phosphate (PLP, pyridoxal 5'-phosphate, P5P) regulation by alteration of Pyridoxamine 5′-phosphate oxidase (PNPO), aka Pyridoxal 5′-Phosphate Synthase. This might well help to explain some of the Adverse Reactions.

https://www.nature.com/articles/s41598-020-70598-7

A useful 2018 review of Tromethamine toxicity in test animals and volunteer healthy Humans with related chemicals intended for use in Cosmetics. Tromethamine administered by Intravenous injection caused a fall in Blood Glucose levels in rats, rabbits, dogs, and Humans. Some evidence of Accumulation in skeletal and Heart muscle, Brain tissue and Cerebrospinal Fluid.

https://www.researchgate.net/publication/325149315_Safety_Assessment_of_Tromethamine_Aminomethyl_Propanediol_and_Aminoethyl_Propanediol_as_Used_in_Cosmetics

A recent paper discussed Hypersensitivity to the Moderna COVID-19 vaccine which also contains Tris. Clever work in Portugal traced the Moderna Anaphylaxis to Tromethamine = Tris. They performed Skin prick and Intradermal tests with excipients or excipient-containing drugs 5 weeks after the first inoculation. They included PEG 3350, PEG 1500 (ROXALL), Polysorbate 20, Methylprednisolone Succinate and acetate, Dexamethasone, Triamcinolone acetonide, Gadobutrol (Gadovist®, contains Tromethamine) and Gadoteric acid (Dotarem®, does not contain Tromethamine).

p4

>>18023087

Only Gadobutrol was positive on intradermal testing, confirming Tromethamine as the Culprit Excipient.

https://www.jiaci.org/ahead-of-print/hypersensitivity-to-the-moderna-covid-19-vaccine-caused-by-tromethamine–peg-is-not-always-the-culprit-excipient

Note that the abstract to this paper is not visible.

The US FDA is fully aware that Tromethamine can induce dangerous side effects, as shown on one supplier website.

The US FDA states, re Tromethamine and pregnant women:

FDA pregnancy category C: Animal reproduction studies have shown an adverse effect on the fetus and there are no adequate and well-controlled studies in humans

https://www.drugs.com/pregnancy/tromethamine.html

Tromethamine is also used in the Ervebo (Ebola Zaire live virus) vaccine. Each 1 ml jab contains 1.21 milligram of the buffer. Common side effects are very similar to those of Pfizer and Moderna Covid19 jabs including: Injection-site reactions (Pain, Swelling, and Redness), Headache, Fever, Muscle Pain, Fatigue, Nausea, Joint Pain and Stiffness = Arthralgia lasting up to 2 years, Rash, Abnormal Sweating, Chills, Paresthesia, Decreases in Lymphocytes and Neutrophils, Anaphylaxis was observed in 2 of 15,399 ERVEBO recipients = 130 per million jabs.

Tromethamine (Trometamol) is used in Dengvaxia Dengue Fever jab.

Australian Government advises it should NEVER be used for primary prevention of initial dengue infection.

Each 0.5 mL dose of Dengvaxia contains 4.5 – 6.0 log10 CCID50 of each of 4 Chimeric Yellow Fever Dengue (CYD) virus serotypes.

Each 0.5 mL dosecontains 2 mg NaCl, 0.56 mg essential amino acids (including L-phenylalanine), 0.2 mg non-essential amino acids, 2.5 mg L-arginine hydrochloride, 18.75 mg Sucrose, 13.75 mg D-trehalose dihydrate, 9.38 mg D-sorbitol, 0.18 mg Trometamol, and 0.63 mg urea.

Serious Adverse Events include: Asthma attack, Urticaria, Convulsions

The Smallpox vaccine approved for use against Monkeypox, Jynneos, is known to cause Anaphyaxis and contains 600 micrograms of Tromethamine per dose.

A recent article discusses other reported Adverse Events and useful history.

Many people injected with Tromethamine report intense Pain.

For Pfizer Covid19 jabs, Pain is reported in Muscles and Joints as well as local reaction with increased effects after each subsequent dose in about 40% of all.

Moderna, which contains more Tromethamine than Pfizer, induces higher levels of Pain with maximum effect after the second dose. The 3rd, Moderna booster dose contains less Tromethamine and less Pain is reported.

Very interesting study from Japan shows that Tromethamine can enhance and prolong Pain sensations by altering Calcium transport via Transient Receptor Potential Vanilloid (TRPV) cation channels.

https://www.researchgate.net/publication/286903571_Tris-hydroxymethyl-aminomethane_enhances_capsaicin-induced_intracellular_Ca2_influx_through_transient_receptor_potential_V1_TRPV1_channels

In 2003 toxicologists at the Chemistry department of the University of Adelaide, collaborating with a pharmacist in Colorado, found that Tromethamine interferes with attempts to study the reaction of carcinogenic Acrolein with adducts at Cysteine, Histidine, and Lysine residues in your body using Western blotting. Providing more clues about mechanisms of Tris damage from Pfizer and Moderna Covid19 jabs.

https://www.researchgate.net/publication/9044782_Reactivity_with_Trishydroxymethylaminomethane_Confounds_Immunodetection_of_Acrolein-Adducted_Proteins

1966 Review of Tromethamine concluded "that there is little current evidence, theoretical, experimental, or clinical, to support the view that Tris has a significant therapeutic role. Some possible exceptions to this general conclusion are considered."

Let’s call for an immediate ban in all jabs.

5 of 5

>>18022855

The WHO is the UN

https://en.wikipedia.org/wiki/List_of_specialized_agencies_of_the_United_Nations

1 Food and Agriculture Organization (FAO)

2 International Civil Aviation Organization (ICAO)

3 International Fund for Agricultural Development (IFAD)

4 International Labour Organization (ILO)

5 International Maritime Organization (IMO)

6 International Monetary Fund (IMF)

7 International Telecommunication Union (ITU)

8 United Nations Educational, Scientific and Cultural Organization (UNESCO)

9 United Nations Industrial Development Organization (UNIDO)

10 Universal Postal Union (UPU)

11 World Bank Group (WBG)

11.1 International Bank for Reconstruction and Development (IBRD)

11.2 International Development Association (IDA)

11.3 International Finance Corporation (IFC)

12 World Health Organization (WHO)

13 World Intellectual Property Organization (WIPO)

14 World Meteorological Organization (WMO)

15 World Tourism Organization (UNWTO)

16 Former specialized agency

17 Related organizations

17.1 Comprehensive Nuclear-Test-Ban Treaty Organization (CTBTO) Preparatory Commission

17.2 International Atomic Energy Agency (IAEA)

17.3 International Organization for Migration (IOM)

17.4 Organisation for the Prohibition of Chemical Weapons (OPCW)

17.5 World Trade Organization (WTO)

End the United States' UN Treaty agreement

>>18023095

Why does RFK appear in front of a highly likely copyrighted Simpsons backdrop containing 'the Alpha and the Omega' & Tesla logo, along with a 'Liquid Death' drink?

Rob Harris

@robharristv

A visual representation of the disaster that is air travel at the moment. A Southwest Airlines rep just said over the intercom that bags are heading to final destination, even if the flight they were scheduled on is cancelled.

https://twitter.com/robharristv/status/1607511661857890304

>>18023183

>>18023185

>>18023189

>>18023188

>>18023223

> 'you end up penniless, the jews do it on purpose'

https://www.thesun.co.uk/news/8713104/michael-jackson-anti-semite-branded-jews-leeches/

>>18023244

Dakhanavar, not unlike the Kardashians, Lisa Barsoomian, etc…

>>18023252

you asked for sauce, anon delivered. the rest is your issue

>>18023264

>>18023275

>>18023299

>Tromethamine

"Pregnancy: Teratogenic Effects: Pregnancy Category C. Ketorolac tromethamine, administered during organogenesis, was not teratogenic in rabbits or rats at oral doses up to 109 times and 303 times the maximum recommended human topical ophthalmic dose, respectively, on a mg/kg basis assuming 100% absorption in humans and animals. When administered to ratsafter Day 17 of gestationat oral doses up to 45 times the maximum recommended human topical ophthalmic dose, respectively, on a mg/kg basis assuming 100% absorption in humans and animals, ketorolac tromethamine resulted in dystocia andincreased pup mortality."

https://dailymed.nlm.nih.gov/dailymed/fda/fdaDrugXsl.cfm?setid=b5f5bdb5-2a0e-437e-a6da-aa262cdc9b89

>>18023312

>seems like a nice man.

sorry fren, you are 'spellbound'. See Magic Johnson and Loop Capital. Also consider what it takes to be and stay famous…

>>18023324

>It is likely a point of desperation determined by

there knowing that they, their family members, friends, and children have been euthanized, and are all essentially walking dead with ADEs/VAIDs, and that there is a global food, fertilizer, and resource shortage/trade stoppage to compound the stress and expedite their SADs

>>18023407

medias purpose is to build trust via pied pipers, then cast reality through them. Every demographic targeted has veery specific roles filled in this regard. Magic has been one for a long while.

There are thousands of talented professional athletic careers, but a few tens of long term broadcasters and spokespersons. Most of whom are most likely MK'd to a degree, initiates and practitioners, or vile fiends with dark deeds.

Magic launched HIV out of the gay-umbrella to the world stage, at the height of his fame. This was not a coincidence, nor was his connection to LA/Hollywood…

A witch's maiden, aka tool of the sorcerers

>>18023430

>Pimps

>>18023517

>>18023498