Tyb
Tom Renz
@TomRenz
Here’s our next cancer article. We are sharing info and while parasite theory is considered “fringe” I’m not sure how we dismiss it entirely in light of the demonstrated effectiveness for #ivermectin and #fenbendazole in some cancer cases. Real science considers ALL theories and then tests them. I personally think we need to do further study to understand the when’s how’s and why’s related to the recognized impact of antiparasitics in cancer and this article provides the background. #Truth #Cancer #MAGA
Cancer and Parasites
As a compendium to my previous substack on cancer, I offer you a brief overview of the theory that cancer is parasitic in origin. Ultimately I believe theory may apply in some cases but not all. Further,…
https://tomrenz.substack.com/p/cancerparasites-ready-for-review
Apr 06, 2024, 9:46 PM
https://truthsocial.com/@TomRenz/posts/112227412465505075
Cancer and Parasites
TOM RENZ
APR 06, 2024
As a compendium to my previous substack on cancer, I offer you a brief overview of the theory that cancer is parasitic in origin. Ultimately I believe theory may apply in some cases but not all. Further, since everything big pharma and mainstream medicine summarily dismisses this theory I think it should be formally studied and proved or disproved so we know. This is not intended to be a comprehensive detailing, it’s simply an introduction. Affording thereto, it’s not possible to cover every expert so if I didn’t include something you believe is relevant - leave me a comment below.
In the mainstream, parasite infection is a commonly disregarded cancer risk factor even though research shows a feasible link between parasites and the development of cancer. Despite the relationship between parasites and other disease, including cancer this theory is often dismissed by ‘big medicine’. Some experts speculate that the reason for this rebuff is clear: money. Simply put, there is no money to be made in a medical system that exists in a parasite paradigm versus a ‘virus’ paradigm. Anti-parasitic medications like Ivermectin and Fenbendazole are pennies on the dollar compared to costly alternatives like chemotherapy and radiation. One treatment option buys the doctor a summer home - while the other does not. The financial incentives to keep such a revelation from being endorsed -or even acknowledged by the system is in the billions, if not more. Their entire industry could topple if people were to learn that the source of their disease could be so simple.
The Center for Disease Controls defines parasite as “an organism that lives on or in a host organism and gets its food from or at the expense of its host. They thrive off of foods such as dairy products, sugar and protein. They list three main classes of parasites that can cause disease in humans: protozoa, helminths, and ectoparasites.
Cancer is characterized by the unchecked growth of aberrant and altered cells that have the capacity to invade nearby tissues. Which, according to some experts is exactly what some parasites do within the body.
Dr. Lee Merritt has served on the Board of the Arizona Medical Association and is a past president of the Association of American Physicians and Surgeons (AAPS). She proposes, in the simplest way I can summarize it - that parasites in the body slowly increase in number over a lifetime and that they can lay thousands, upon thousands of eggs a day and form intracellular cysts. As more cysts present themselves it takes more of your immune system to keep them in check. The more active the immune system, the more likely you are to experience autoimmune disease. She postulates that when their numbers get too many, when these microscopic invaders are out of control and egg sacs enlarge and invade nearby tissues- this is when we see metastasis. Metastasis is when cancer has spread to a different part of the body part than where it started. Cancer spreads this way via the lymphatic system and it’s plausible to picture these invaders traveling throughout the body wreaking havoc.
In 1890 Scottish microbiologist and pathologist William Russell reported finding parasitic spores within cancer cells. He published his findings in the British Medical Journal and went on to formulate a comprehensive hypothesis explaining how cancer is a parasitic disease. As previously mentioned, these theories have routinely been dismissed within the medical community which remains steadfast in the ‘virus’ paradigm.

I recommend you read ‘How Parasites Get Their Hooks into Human Cells’, published by the Whitehead Institute for biomedical research. It explains the chain of cellular events that see parasites replicate and travel within cells and how other factors like calcium play a role.
“Whitehead Institute Member Sebastian Lourido studies apicomplexan biology using the species Toxoplasma gondii (T. gondii), which causes toxoplasmosis, a disease that can be deadly in people with compromised immune systems.
Active T. gondii parasites transition between two modes: replicating inside of one host cell and spreading to new cells. The transition between these modes happens very quickly: parasites will be busy replicating and then will rapidly exit the host cell and travel to new cells to invade them. Once inside a new cell, the parasites can switch back to reproducing or enter into a chronic stage—a hardy, inert form that can hide from the host’s immune system indefinitely.
Researchers knew that T. gondii starts its transition from the reproductive to the spreading mode with a calcium signal within the parasite. Calcium is an important signal that many cells use to trigger the quick release of different molecules, such as neurotransmitters from neurons and insulin from pancreatic cells in humans. In T. gondii, calcium is the signal that triggers release of molecules parasites need to exit host cells, travel, and invade. These include molecules to make the host cell membrane permeable and sticky molecules that the parasite uses to attach to and glide along host tissue, enabling it to move in the environment outside of the host cell. These molecules are packaged in small membrane-bound containers called micronemes, hundreds of which are scattered throughout the front third of the parasite cell.
The calcium signal begins a chain of signaling, like a game of telephone, that ultimately triggers the parasite cell to release the contents of these microneme packages. The cell does this by attaching the packages to a cellular conveyor belt system that runs along microtubules, part of the cytoskeleton. This whizzes them to the very front of the parasite, where their contents can be released outside of the parasite through a process called exocytosis, in which the membrane of the package fuses with the membrane of the parasite cell.”
A review published in Science Direct “Helminths in Human Carcinogenesis" reveals the following:
“Johanes Fibiger won the only Nobel Prize for helminthology in 1926 for the induction of gastric cancer in rats by feeding them cockroaches infected with Spiroptera neoplastica larvae (Campbell, 1997). The idea of parasitic worms causing cancer was not new. Fibiger had been aware of the causal relationship of Schistosoma haematobium and human urinary bladder cancer. His dissection of wild rats had yielded remarkable finding—stomach tumours containing worms.
Fibiger's worm, S. neoplastica (later renamed Gongylonema neoplastica), provided a unique scientific tool for the experimental induction of cancer in mammalian hosts. Fibiger's work was later criticized for the gastric tumours not being true cancers, but merely worm‐induced hyperplasia associated with vitamin A deficiency. Today, the International Agency for Research on Cancer (IARC) has in part vindicated Fibiger's belief in parasite‐induced cancer by labelling S. haematobium and Opisthorchis viverrini as definitely carcinogenic (group 1) and Clonorchis sinensis as probably carcinogenic (group 2) to humans (IARC, 1994).”
In November 2015, a study published in the New England Journal of Medicine called ‘Malignant Transformation of Hymenolepis nana in a Human Host’, revealed a shocking case in which cancer developed into a different kind of infectious parasite as a result of a tapeworm infection. Researchers found tapeworm DNA in the cancer cells when they looked at the immunocompromised patient's lymph node tissue.
“The problem of proving the role of parasites in cancer induction is difficult because of their complex natural histories and long asymptomatic latent periods during which numerous endogenous and exogenous factors can interact to obfuscate causality.”
Herrera and Ostroskyu2010Wegman, 2001
If youu2019re thinking itu2019s only as deep as u2018people with compromised immune systems are just more susceptible to parasitesu2019 - thatu2019s fair. After all, correlation does not necessarily equal causation, however, it has been demonstrated that the tick-borne parasite T. parva creates a disease that is almost exactly like lymphoma, and that inoculating mice with the C. parvum parasite causes colon cancer abruptly.
Additionally, the u201cHelminths in Human Carcinogenesis" reveals the following:
u201cThe International Agency for Research on Cancer (IARC) considered three species of trematodes, Schistosoma haematobium, Opisthorchis viverrini and Clonorchis sinensis, responsible for helminth induced human cancer; they considered S. haematobium and O. viverrini as group 1 carcinogens and C. sinensis as a group 2 carcinogen [2].
Helminth infections are of great importance globally with countless millions of humans being infected or at risk of infection. Mathematical models have been used to calculate the risk of cancer due to infection; the calculated risk factor indicates that about 15% of all cases globally can be attributed to infections including those due to schistosomes and liver flukes. The infectious origin of a cancer implies that it is preventable. Therefore, if infections were prevented by increased educational efforts and improved public health initiatives, there would be considerably fewer cases of cancer in both developed and underdeveloped countries.u201d
The Lancet reports:
u201cAmong parasitic diseases, infections with the two fish-borne liver flukes of the family Opisthorchiidae (trematodes), specifically Opisthorchis viverrini and Clonorchis sinensis, can induce cholangiocarcinoma, and infection with the blood fluke Schistosoma haematobium may cause cancer of the urinary bladder (Bouvard et al., 2009).u201d
Knowing this, itu2019s not a stretch to come to the conclusion that ridding the body of parasites could effectively prevent or treat some cancer. Many of us have heard about the Joe Tippens Protocol, where a man allegedly cured his late-stage cancer with an inexpensive veterinary product, a de-wormer called Fenbendazole. Experts like Dr. Bryan Ardis have weighed in on the discussion and concluded that anti parasitic drugs are an effective treatment with a documented history of success. He explains that approximately 50% of the cancerous tumors/polyps he saw with his patients had been misdiagnosed and were actually parasitic egg sacs. He indicated that 70% of autoimmune cases seen in his practice - were caused by parasites.
ufffc
In a 2021 Johns Hopkins study published in the journal Oncotarget on July 6, Gregory Riggins, M.D., Ph.D., professor of neurosurgery and oncology at the Johns Hopkins University School of Medicine, and his team u2018used two different mouse models to determine that the anti-parasitic drug mebendazole could slow or stop the growth and spread of both early and late-stage pancreatic cancer. Riggins and his team administered mebendazole to mice that were genetically engineered to develop pancreatic canceru2019. Read that again: they genetically manipulated them to develop cancer.
Riggins concluded: u201cWe think that mebendazole could have a role in all stages. It was particularly effective for pancreatic cancer that was detected early.u201d
Oncologist Dr. William Makis has compiled data that shows that u2018Fenbendazole has at least 12 proven anti-cancer mechanisms in vitro and in vivou2019. I encourage you to read his article where he goes in depth on each of the studies that support the following conclusions of anti-cancer mechanisms.
ufffc
There is evidence to suggest that anti-parasitic medications treat the following cancers:
ufffc
I will tell you simply that I do not believe that anything here conclusively proves that all cancer is parasitic but the questions are fair and legitimate. I also do not believe that FenBen, Ivermectin or anything else will be the magic pill that cures everything we call cancer. That said, the parasite hypothesis deserves legitimate and unbiased research and while I may not believe those two drugs will cure all cancer I absolutely believe they could have a therapeutic role and potentially cure some cancers. Ultimately I think it is fair to ask whether anti-parasitic drugs are showing promise in fighting some cancer because the cancer is a result of parasites or if it is because the cancer is susceptible to treatments for parasites.
Ultimately anti parasitic drugs should investigated to determine if and how they should be included in the treatment of cancer. Fenbendazole is just ONE of the drugs in this class with others like Ivermectin proving to be equally as promising. The real question is, will Big Pharma or our crooked government ever legitimately investigate this?
"They know that Cancer is Parasites, but they're not letting anyone know because they don't want to lose their funding."
Dr. Lee Merritt
Sources:
-
Parasites that Can Lead to Cancer | American Cancer Society | American Cancer Society
-
brmedj04652-0016.pdf (nih.gov)
-
How Little Do Users Read? (nngroup.com)
-
Abstract 2313: Fenbendazole induces cell cycle arrest in colorectal cancer cells and patient-derived colon cancer organoids | Cancer Research | American Association for Cancer Research (aacrjournals.org)
-
The Overlooked Miracle Drug for Cancer? Why Big Pharma Fears Fenbendazole u2013 Vigilant News Network
-
Joe Tippins' Fenbendazole Protocol For Cancer (deeprootsathome.com)
-
https://rumble.com/v499vvf-parasites-the-cause-of-cancer-and-covid.html
-
https://rumble.com/v493ooo-dr-lee-merritt-on-cancers-and-darpa-mind-control.html
-
https://yournews.com/2023/08/24/2628420/dr-lee-merritt-cancer-tumors-are-parasites/
-
The Medical Rebel (drleemerritt.com)
-
Parasites–a New Paradigm (rumble.com)
-
Parasites: Types, Symptoms, Treatment & Prevention (clevelandclinic.org)
-
How Parasite Infection Can Lead to Cancer (naturalmentor.com)
-
Parasite-Derived Cancer | New England Journal of Medicine (nejm.org)
-
Cancer: An Unbiased Overview In Preparation for Our Upcoming War On Cancer (substack.com)
-
Parasite-microbe-host interactions and cancer risk | PLOS Pathogens
-
Antiparasitic Drugs: Malaria, Toxoplasmosis, Parasitic Infection (clevelandclinic.org)
-
How parasites get their HOOKs into human cells | Whitehead Institute (mit.edu)
-
Helminths in human carcinogenesis - ScienceDirect
-
Project MUSE - The Worm and the Tumor: Reflections on Fibiger's Nobel Prize (jhu.edu)
-
The Dr. Ardis Show (thedrardisshow.com)
VIDEO PROOF: CANCER IS CAUSED BY PARASITES
30:49
https://www.youtube.com/watch?v=70yyHUzq-sU
Cancer is a Microparasitical Infection, and We All Have It
53:02
https://www.youtube.com/watch?v=bcgUBVj9i70
it seems to have killed the diabetes
so
i LOVE taking ivermectin and elemental zinc
ivermectin is a zinc ionisphore, like a high caliber gun, i splash it on, topical hand full
zinc is the bullet, 60mg
7 days
then once a month, only
then 7 days every 4 months
makes me feel healthy and ZERO side effects
NEVER UNDER DOSE with ivermectin..
that only happens the first time you take ivermectin
just ghost it
>>20690882, >>20690903, >>20690909, >>20690911, >>20690912, >>20690917 Tom Renz: Cancer and Parasites, ivermectin fenbendazole
notable
i got lucky, killed the parasites in the pancreas and lost weight
How parasites get their HOOKs into human cells
Written by Greta Friar
October 04, 2023
Apicomplexan parasites are single-celled organisms that infect millions of people a year around the world. Different species of apicomplexans are responsible for spreading malaria, cryptosporidiosis (a severe diarrheal disease that affects young children), and other serious illnesses. Whitehead Institute Member Sebastian Lourido studies apicomplexan biology using the species Toxoplasma gondii (T. gondii), which causes toxoplasmosis, a disease that can be deadly in people with compromised immune systems. Uncovering new aspects of the parasites’ biology could lead to therapies to eliminate them or prevent disease symptoms.
Active T. gondii parasites transition between two modes: replicating inside of one host cell, and spreading to new cells. The transition between these modes happens very quickly: parasites will be busy replicating and then will rapidly exit the host cell and travel to new cells to invade them. Once inside a new cell, the parasites can switch back to reproducing or enter into a chronic stage—a hardy, inert form that can hide from the host’s immune system indefinitely.
Researchers knew that T. gondii starts its transition from the reproductive to the spreading mode with a calcium signal within the parasite, but they did not know much about the chain of cellular events between that first signal and the actual switch in modes. In research published in the journal eLife on November 7, Lourido, former graduate student in his lab Alex Chan, and colleagues shed light on what happens inside of the parasite cell to enable this transition.
Piecing together the chain of events
Calcium is an important signal that many cells use to trigger the quick release of different molecules, such as neurotransmitters from neurons and insulin from pancreatic cells in humans. In T. gondii, calcium is the signal that triggers release of molecules parasites need to exit host cells, travel, and invade. These include molecules to make the host cell membrane permeable and sticky molecules that the parasite uses to attach to and glide along host tissue, enabling it to move in the environment outside of the host cell. These molecules are packaged in small membrane-bound containers called micronemes, hundreds of which are scattered throughout the front third of the parasite cell.
The calcium signal begins a chain of signaling, like a game of telephone, that ultimately triggers the parasite cell to release the contents of these microneme packages. The cell does this by attaching the packages to a cellular conveyor belt system that runs along microtubules, part of the cytoskeleton. This whizzes them to the very front of the parasite, where their contents can be released outside of the parasite through a process called exocytosis, in which the membrane of the package fuses with the membrane of the parasite cell.
When T. gondii parasites get the signal to exit the host cell and travel, the micronemes (in yellow) get quickly shuttled to the very tip of the parasite cell.
u201cParasites need to control how and when they interact with host cells strategically, in order to survive in a hostile environment,u201d says Lourido, who is also an associate professor of biology at the Massachusetts Institute of Technology. u201cThey need to release the contents of micronemes precisely at the moment when theyu2019re undertaking the transition from replication to movement, and so this transition happens incredibly quickly. How they achieve that, at a cellular and molecular level, has remained quite mysterious.u201d
What Lourido and Chan wanted to understand was what happens between the calcium signal and the release of the microneme contents. What cellular players are participating in this game of telephone that enables parasites to quickly transition modes? Lourido had helped to identify the next step in the process when he was a graduate student: the calcium signal activates a molecule called calcium-dependent protein kinase 1 (CDPK1). He found that CDPK1 is necessary for exocytosis, and so for the parasites to exit the host cell and travel. CDPK1 is not found in humans, which makes it a good possible drug target, as inhibiting it would only affect the parasite and not the host.
Now, using newer technologies, Chan developed an approach to identify the molecules that CDPK1 acts onu2014the next players in the game of telephoneu2014helping researchers better understand the role of CDPK1 and gain a more complete picture of how T. gondii switches into spreading mode. CDPK1u2019s function is to attach a molecular structure called a phosphate group to other molecules. Chan identified 163 proteins that receive a phosphate group when CDPK1 is activated, some of which are likely to play roles in exocytosis. Chan also figured out which of the proteins CDPK1 targets directly, versus those that receive a phosphate group from another intermediary. Understanding what molecules CDPK1 targets, and what they do, will help researchers understand how a drug to inhibit CDPK1 would actually affect cells.
Narrowing in on a key player
One of the molecules that the researchers found to be acted on directly by CDPK1, a previously unidentified protein, caught Chanu2019s attention. Based on its similarity to molecules found in other species, Chan suspected that it was an activating adaptor: a molecule that helps connect cargo like micronemes to the cellu2019s conveyor belt and moves them along it. Chan named the molecule HOOK, after the similar molecules in other species. He also identified three key proteins that work with HOOK.
When Chan removed or deactivated HOOK, T. gondii parasites were still able to exit the host cell but were unable to travel or invade, compromising infection. This is consistent with the idea that HOOK is needed to move micronemes along the cellu2019s conveyor belt. Before the parasite receives the signal to switch modes, it already has a few micronemes locked and loaded at its tip, ready to release their contents outside of itself. Emptying these micronemes provides enough material to escape the host cell. However, without HOOK to help shuttle more micronemes to the cell tip, the parasite quickly runs out of material. T. gondii need to constantly release the sticky molecules found inside of micronemes as they move, almost like they are building a road as they travel along it. With no more micronemes, thereu2019s no more road. The conveyor belt system that HOOK helps to activate is very good at rapidly and continuously shuttling materials forward to keep building new road.
The list of CDPK1 targets that the researchers compiled may contain other players that contribute to other aspects of the parasiteu2019s transition between modes, but HOOK is clearly a major player, necessary for the parasite to succeed at traveling to new host cells. Chan and Louridou2019s findings not only shed light on how parasites transition so quickly between modes, but could also provide new insights into how many species, including humans, move cargo inside of cells, as these species use similar cellular machinery to that found in T. gondii.
u201cOur work to understand how micronemes get shuttled across the cell really benefited from reading about work done on similar cargo trafficking in other species,u201d Chan says. u201cThere are a lot of unanswered questions about how this sort of trafficking is regulated, and now I think T. gondii could be a good model to understand this more broadly.u201d
Notes
Alex W Chan, Malgorzata Broncel, Eden Yifrach, Nicole Haseley, Sundeep Chakladar, Elena Andree, Alice L Herneisen, Emily Shortt, Moritz Treeck, Sebastian Lourido (2023). "Analysis of CDPK1 targets identifies a trafficking adaptor complex that regulates microneme exocytosis in Toxoplasma." eLife 12:RP85654. https://doi.org/10.7554/eLife.85654.3
https://wi.mit.edu/news/how-parasites-get-their-hooks-human-cells
Ivermectin has cured me of a number of issues.
I have 3 liters of the stuff on hand at all times
And bottles of elemental zinc
it is very easy to find
Ivermax Pour-On (B)
This page contains information on Ivermax Pour-On (B) for veterinary use.
The information provided typically includes the following:
Ivermax Pour-On (B) Indications
Warnings and cautions for Ivermax Pour-On (B)
Direction and dosage information for Ivermax Pour-On (B)
Ivermax Pour-On (B)
This treatment applies to the following species:
Beef Cattle
Company: Aspen
(ivermectin topical solution)
POUR-ON FOR CATTLE
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Contains 5 mg ivermectin/mL
https://www.drugs.com/vet/ivermax-pour-on-b.html
you made look like cryton
you made him look like cryton*
>Pour on isn't the same as internal
how so? you uneducated dipshit
do your own investigation and experiments
Series: A contagious collection: infectious disease research
Enjoy this collection of stories on Whitehead Institute’s recent infectious disease research, from apicomplexan parasites to zika virus—just remember to wash your hands well after reading.
Parasites in white, hlue, and magenta
Pathogens including viruses, bacteria, and parasites are all around us. They are humanity’s frequent foes, and the fight against them has led to many innovations in public health. They are also fascinating sources of lessons about biology, including our own. Pathogens must survive in extremely hostile conditions—human bodies that want to destroy them—and they have evolved ingenious solutions to stay alive. They can also co-opt their hosts’ biology to turn it from hostile to helpful, using human cells to make more copies of themselves, something many pathogens cannot do on their own. Explore the stories below to learn more about some captivating pathogens, the diseases they cause, and how Whitehead Institute research could help lead to prevention strategies, treatments, and cures.
Anticipating and countering infectious diseases
Climate Change
February 28, 2023
Climate change and economic development are increasing our risk of being infected by parasites and harmful viruses and bacteria. Whitehead Institute researchers are leveraging their expertise to better assess where pathogens will spread and evaluate their risk to humans.
Parasites
Image
Micrograph: an oval shape containing two others, each outlined in a grid of purple lines and with a bright top
How parasites get their HOOKs into human cells
Infectious disease
October 04, 2023
Whitehead Institute Member Sebastian Lourido and colleagues identified key parts of the pathway that enables apicomplexan parasites to exit one host cell, and travel to infect others, including the important role of a molecule they called HOOK. This finding illuminates how signals can move in cells and could inform drug development against the parasites.
Image
Micrograph of a green, oval parasite with tip pressed against the edge of a cell
CLAMP complex helps parasites enter human cells
Infectious disease
October 27, 2023
Apicomplexan parasites are responsible for several prevalent infectious diseases including malaria. Whitehead Institute Member Sebastian Lourido and colleagues identified a protein complex that the parasites require for a critical step in invading host cells.
Image
Left: clusters of magenta ovals. Right: ovals now encased in larger green shape
Positive feedback loop drives transition of Toxoplasma gondii to chronic stage
Infectious disease
April 20, 2023
Research from Whitehead Institute Member Sebastian Lourido finds an RNA-binding protein that is key to helping the parasite Toxoplasma gondii commit to its dormant stage.
Image
Images of toxoplasma parasites inside of a host cell
Multimedia feature: A "model" parasite
Infectious disease
August 24, 2019
Whitehead Institute researchers unravel the unique biology of apicomplexans — the parasites responsible for malaria, toxoplasmosis, and other diseases impacting global health.
Image
Close up of Sebastian Lourido
Audio feature: Sebastian Lourido discusses his lab's work on Toxoplasma gondii
Infectious disease
March 07, 2024
In a brief audio clip, Sebastian Lourido discusses his lab's recent work.
Viruses
Image
Micrograph of a neuron in green with blue nucleus, blue stained cells with red dots in background
SARS-Cov-2, the virus behind Covid-19, can infect sensory neurons
Covid-19
September 05, 2023
Whitehead Institute Founding Member Rudolf Jaenisch and colleagues found that SARS-Cov-2 can infect sensory neurons and alter their gene expression. This may help to explain the peripheral nervous system symptoms associated with Covid-19, such as loss of smell.
Image
Blue circles with red rims
New research supports finding explaining why some patients may test positive for COVID-19 long after recovery
Covid-19
February 28, 2023
New research from Whitehead Institute Founding Member Rudolf Jaenisch’s lab adds evidence to their finding that SARS-CoV-2 can integrate into the human genome, but finds that a model of the mRNA vaccine does not integrate.
Image
An illustration of a two-headed scientist, one head looking at a microscope and the other listening to a patient's chest with a stethoscope.
When the blueprint shifts: research at Whitehead Institute into developmental disorders, diseases, and differences
Development and regeneration
July 25, 2022
Whitehead Institute research projects continue to provide important insights into human biology, and they are paving the way for future treatments to address developmental disorders and diseases.
https://wi.mit.edu/series/contagious-collection-infectious-disease-research
Devin Nunes
@DevinNunes
Don’t miss @JackPosobiec 
rumble.com/v4nhpnt-unhumans-th
https://truthsocial.com/@DevinNunes/posts/112227835446926079
Unhumans: The Secret History of Communist Revolutions with guest Jack Posobiec
53:48
https://rumble.com/v4nhpnt-unhumans-the-secret-history-of-communist-revolutions-with-guest-jack-posobi.html
https://rumble.com/embed/v4kwhye/?pub=4
Wholesome.
https://truthsocial.com/@SteakforBreakfast/posts/112227721419418241
Donald J. Trump
@realDonaldTrump
Crooked Judge Juan Merchan is not allowing me to talk, is taking away my First Amendment Rights, he’s got me GAGGED, because he doesn’t want the FACTS behind the Gag to come out. How many Corrupt, Biased, Crooked Joe Biden-“Protection Agency” New York Judges do I have to endure before somebody steps in? I had New York Federal Judge, Lewis Kaplan, with a woman who I never knew, and had nothing to do with, until she sued me for “defamation.” She did not know what day, month, or year the supposed “incident” took place - She knew nothing. Kaplan, a Hillary friend, wouldn’t even let my lawyers put on a proper case, made it two cases instead of one, took away my American Right to defend myself, and was a Crazed Bully as his wife and friends sat in the Courthouse, every day in their little roped off section, and prodded him on in awe. This mysterious lady, who said rape is sexy, got $91 Million Dollars.
Apr 06, 2024, 1:06 PM
https://truthsocial.com/@realDonaldTrump/posts/112225366117672509
Donald J. Trump
@realDonaldTrump
Page 2: Then, of course, we have Arthur Engoron, a total lunatic, whack job, who ruled that I should pay an almost $500 Million Dollar “fine” for having done nothing wrong (No Victims, No Damages, Ironclad Disclaimer, ONLY SUCCESS!), in fact, he was the one who committed fraud when he purposely, at the direction of Racist, Corrupt A.G. Letitia James, undervalued Mar-a-Lago at $18 Million Dollars.
Apr 06, 2024, 1:06 PM
https://truthsocial.com/@realDonaldTrump/posts/112225365104178478
Donald J. Trump
@realDonaldTrump
Page 3: Now, we have Merchan, who is not allowing me to talk, thereby violating the Law and the Constitution, all at once. It is so bad what he is trying to get away with - How was he even chosen for this case??? I heard he fought like hell to get it, and all of the rest of them also! If this Partisan Hack wants to put me in the “clink” for speaking the open and obvious TRUTH, I will gladly become a Modern Day Nelson Mandela - It will be my GREAT HONOR. We have to Save our Country from these Political Operatives masquerading as Prosecutors and Judges, and I am willing to sacrifice my Freedom for that worthy cause. We are a Failing Nation, but on November 5th, we will become a Great Nation again. MAKE AMERICA GREAT AGAIN!
Apr 06, 2024, 1:05 PM
https://truthsocial.com/@realDonaldTrump/posts/112225363872196974
any word on an antiparasitical that cures muscle dystrophy?
AMERICAN NEWS Apr 5, 2024
Jack Smith may seek to remove Trump-appointed Judge Aileen Cannon from Mar-a-Lago seized docs case
"Deranged 'Special' Counsel Jack Smith … should be sanctioned or censured for the way he is attacking a highly respected Judge, Aileen Cannon, who is presiding over his FAKE Documents Hoax case in Florida."
Special counsel Jack Smith may seek the recusal of the Trump-appointed judge presiding over the case regarding classified documents.
According to NBC News, Smith recently criticized Judge Aileen Cannon's handling of Trump's “fundamentally flawed” claims regarding official and personal records when she asked the prosecution and the defense to bring forward competing versions of instructions for jurors in the case. Smith went on to indicate that if Cannon ruled against federal prosecutors, he could appeal to have her removed from the case.
Palm Beach County State Attorney David Aronberg explained that Smith is “close to pushing the nuclear button,” and Cannon’s proposed jury instructions may have “pushed him to the breaking point.” The jury instruction proposal would consider the Trump team’s interpretations of how former Presidents can preserve classified documents after leaving office.
Cannon has stated that this decision was nothing more than “a genuine attempt… to better understand the parties’ competing positions and the questions to be submitted to the jury in this complex case of first impression.” She also called Smith’s request “unjust” for a ruling on whether the legal premise behind her request is a “correct formulation of the law,” per NBC News.
Trump's legal team has argued that the Presidential Records Act grants Trump the authority to determine the classification status of records, including those seized by investigators at his Mar-a-Lago estate. However, Smith's office has challenged this defense, asserting that it lacks factual basis and casting doubt on Trump's claim that the documents were made personal during his presidency.
In response to Smith's criticisms against the judge, Trump took to Truth Social to defend Judge Cannon, suggesting that Smith should face sanctions or censure for his remarks regarding the judge's request for jury instruction proposals.
"Deranged 'Special' Counsel Jack Smith, who has a long record of failure as a prosecutor, including a unanimous decision against him in the U.S. Supreme Court, should be sanctioned or censured for the way he is attacking a highly respected Judge, Aileen Cannon, who is presiding over his FAKE Documents Hoax case in Florida," Trump said in his post.
Smith's office has also expressed frustration with the pace of proceedings under Judge Cannon, particularly as Trump seeks additional time to present his arguments in Florida.
https://thepostmillennial.com/jack-smith-may-seek-to-remove-trump-appointed-judge-aileen-cannon-from-mar-a-lago-seized-docs-case?utm_campaign=64501
Donald Trump raises over $50.5 MILLION at Palm Beach dinner, smashing records
"Today, the Trump campaign and Republican National Committee (RNC) announced that President Trump’s Palm Beach dinner will raise over $50.5 million." The amount is double that of President Joe Biden's recent NYC fundraiser.
President Donald Trump and groups behind him smashed fundraising records at a West Palm Beach fundraiser dinner on Saturday. The amount was double that of President Joe Biden's recent NYC fundraiser.
In a press release from the Republican National Committee, the announcement was made about the record-breaking fundraiser.
"Today, the Trump campaign and Republican National Committee (RNC) announced that President Trump’s Palm Beach dinner will raise over $50.5 million – smashing every fundraising record in history for a Republican or Democrat. This comes after Biden’s desperate New York event took three presidents to raise just $25 million, and as the Trump campaign and RNC’s small dollar fundraising program continues to grow each month," the release stated.
Trump campaign senior advisors Chris LaCivita and Susie Wiles said, “Tonight will be an incredible night for President Trump and the Republican Party, raising an astounding $50.5 million."
They added, “Meanwhile, after locking up the nomination in one of the fastest primaries in modern political history, Donald J. Trump is winning poll after poll and proving that the enthusiasm is on his side. It’s clearer than ever that we have the message, the operation, and the money to propel President Trump to victory on November 5.”
RNC Chairman Michael Whatley and Co-Chairman Lara Trump spoke to what Americans have perceived as Biden’s largest failures in the polls, namely the border and the economy.
“The Republican Party is united behind the effort to elect President Donald J. Trump, and Americans are lining up to join our movement and retire Crooked Joe Biden once and for all,” they added.
Trump reacted to amount raised at the event on Truth Social as well.

In a statement made prior to the fundraiser at Mar-a-Lago, Trump said that the upcoming 2024 election will be the most important … election we’ve ever had.”
https://thepostmillennial.com/breaking-donald-trump-raises-50-5-million-at-palm-beach-dinner-smashing-records
i saw a ufo today
looked straight up
it was a large white light object, a plane was flying above it in the opposite direction
nice
>>20691263