Gut bacteria can cause/cure Tinnitus
The most consistently and scientifically associated gut bacteria with tinnitus is Blautia, particularly its reduced abundance in affected individuals. Below is a detailed, evidence-based explanation:
✅ Key Findings From Peer-Reviewed Studies:
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Reduced Blautia in Tinnitus Patients
Study: Zhao et al. (2021), Frontiers in Cellular and Infection Microbiology
Method: 16S rRNA sequencing of gut microbiota from tinnitus patients and controls.
Finding:
Blautia and Eubacterium hallii group were significantly reduced in chronic subjective tinnitus patients.
Blautia is a butyrate-producing genus, which supports gut barrier integrity and modulates systemic inflammation.
Depletion may contribute to neuroinflammatory processes implicated in tinnitus.
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Tinnitus Severity Correlated With Gut Dysbiosis
Study: Nagashima et al. (2022), Scientific Reports
Finding: Patients with more severe tinnitus showed alterations in the abundance of several genera, with particular reductions in Blautia and Faecalibacterium.
🔬 Mechanistic Implications:
Blautia and Butyrate:
Blautia produces butyrate, a short-chain fatty acid (SCFA) with anti-inflammatory and neuroprotective effects.
Butyrate can cross the blood-brain barrier and modulate microglial activation, potentially reducing central sensitization linked to tinnitus.
Neuroinflammation Link:
Chronic tinnitus is thought to involve neuroinflammation, particularly in auditory and limbic brain regions.
A gut microbiome depleted in Blautia may exacerbate systemic inflammation and weaken the gut-brain barrier.
🔬 Other Notable Microbiota Trends (Less Consistent):
Increased: Bacteroides, Ruminococcus gnavus group (pro-inflammatory species).
Decreased: Faecalibacterium, another butyrate producer, often depleted in neuroinflammatory and psychiatric conditions.
🎯 Summary — Direct Answer:
The gut bacterium most scientifically and consistently associated with tinnitus is Blautia, particularly its reduced abundance. This change is likely tied to decreased butyrate production and increased systemic and neuroinflammation, which are implicated in tinnitus pathophysiology.
God Bless.