Anonymous ID: 30f01b June 3, 2025, 2:33 p.m. No.23118438   ๐Ÿ—„๏ธ.is ๐Ÿ”—kun   >>8441 >>8458 >>8466 >>8518 >>8596 >>8696 >>8747 >>9058

T-2 is a fungus

 

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JUST IN: U.S. authorities have stopped a plot by two Chinese agents to smuggle a toxic fungus into a Michigan lab that could have wreaked havoc on the entirety of Americaโ€™s food production capabilities.

https://x.com/outbreakupdates/status/1930011543862661410

 

T-2 mycotoxin is a trichothecene mycotoxin. It is a naturally occurring mold byproduct of Fusarium spp. fungus which is toxic to humans and other animals. The clinical condition it causes is alimentary toxic aleukia and a host of symptoms related to organs as diverse as the skin, airway, and stomach. Ingestion may come from consumption of moldy whole grains. T-2 can be absorbed through human skin.[2] Although no significant systemic effects are expected after dermal contact in normal agricultural or residential environments, local skin effects can not be excluded. Hence, skin contact with T-2 should be limited.

 

History

Alimentary toxic aleukia (ATA), a disease which is caused by trichothecenes like T-2 mycotoxin, killed many thousands of USSR citizens in the Orenburg District in the 1940s. It was reported that the mortality rate was 10% of the entire population in that area. During the 1970s it was proposed that the consumption of contaminated food was the cause of this mass poisoning. Because of World War II, harvesting of grains was delayed and food was scarce in Russia. This resulted in the consumption of grain that was contaminated with Fusarium molds, which produce T-2 mycotoxin.[3]

 

In 1981, the United States Secretary of State Alexander Haig and his successor George P. Shultz accused the Soviet Union of using T-2 mycotoxin as a chemical weapon known as "yellow rain" in Laos (1975โ€“81), Kampuchea (1979โ€“81), and Afghanistan (1979โ€“81), where it allegedly caused thousands of casualties.[4] Although several US chemical weapons experts claim to have identified "yellow rain" samples from Laos as trichothecenes, other experts believe that this exposure was due to naturally occurring T-2 mycotoxin in contaminated foods.[5] Another alternative theory was developed by Harvard biologist Matthew Meselson, who proposed that the "yellow rain" found in Southeast Asia originated from the excrement of jungle bees.[6] The first indication for this theory came from finding high levels of pollen in the collected samples, giving the substance its yellow color. It was also found that jungle bees in this area fly collectively in great numbers, at altitudes too high to be easily seen, producing showers of feces that could have been mistaken for sprays from aircraft.[7] Further testing later determined that the oily liquid was, in fact, the pollen-filled feces of jungle bees.

https://en.wikipedia.org/wiki/T-2_mycotoxin#:~:text=T-2%

Anonymous ID: 30f01b June 3, 2025, 2:44 p.m. No.23118472   ๐Ÿ—„๏ธ.is ๐Ÿ”—kun   >>8598

>>23118458

I saw an ant commit suicide in a river, it walked in, held onto a grain of sand until it released an air bubble, then it died and floated away.

Same thought about something you don't see every day.

I still think about that ant and what could have been so terrible in its life.

Anonymous ID: 30f01b June 3, 2025, 3:27 p.m. No.23118596   ๐Ÿ—„๏ธ.is ๐Ÿ”—kun   >>8747 >>9058

>>23118518

>>23118438

T-2 Warningdrop solved Jun 30 2021

 

Jun 30th 2021

@Huskydog Good, the more they start eating each other, the more the people will seeโ€ฆ

 

So it would seem that this so called "RARE" clotting disorder, isn't so rare at all.

 

BTW, I did some research into what Q mentioned, being "T-2". I had no idea what it was until I started researching and came across a document describing "T-2 Toxin"

 

Turns out it's a fungus, which when combined with ethyl-glycol as used in vaccines, messes with blood clotting and becomes very toxic. As it turns out, the vaccine also uses a fungus, as a binding agent. I don't have a direct connection, but it does seem very coincidental that the same ingredients are in the vaccine which causes blood clotting problems and kills peopleโ€ฆ,

 

But more importantly, it's commonly used as a BIOWEAPONโ€ฆ

 

T-2 Mycotoxin - Toxins as Biochemical Weapons

I can't post the link to it here for some reason?

https://gab.com/JohnnyBeBad1/posts/106499370471710534

Anonymous ID: 30f01b June 3, 2025, 3:37 p.m. No.23118625   ๐Ÿ—„๏ธ.is ๐Ÿ”—kun   >>8635

>>23118503

>https://abcnews.go.com/Politics/2-chinese-nationals-charged-smuggling-potential-agroterrorism-fungus/story?id=122454213

 

Turns out it's a fungus, which when combined with ethyl-glycol as used in vaccines, messes with blood clotting and becomes very toxic. As it turns out, the vaccine also uses a fungus, as a binding agent. I don't have a direct connection, but it does seem very coincidental that the same ingrediants are in the vaccine which causes blood clotting problems and kills peopleโ€ฆ,

 

But more importantly, it's commonly used as a BIOWEAPONโ€ฆ

 

T-2 Mycotoxin - Toxins as Biochemical Weapons

Anonymous ID: 30f01b June 3, 2025, 4:01 p.m. No.23118696   ๐Ÿ—„๏ธ.is ๐Ÿ”—kun   >>8708 >>8711 >>8747 >>9058

>>23118438

moarT-2 Fungus

 

Mechanism of action

The toxicity of T-2 toxin is due to its 12,13-epoxy ring.[14] Epoxides are in general toxic compounds; these react with nucleophiles and then undergo further enzymatic reactions. The reactivity of epoxides can lead to reactions with endogenous compounds and cellular constituents like DNA bases and proteins.[15] These reactions could be the reason for the noticed actions and effects of T-2 mycotoxin. The toxic compound influences the metabolism of membrane phospholipids, leads to an increase of liver lipid peroxidases and has an inhibiting effect on DNA and RNA synthesis. In addition it can bind to an integral part of the 60s ribosomal subunit, peptidyltransferase, thereby inhibiting protein synthesis. These effects are thought to be the explanation for T-2 toxin inducing apoptosis (cell death) in different tissues as the immune system, the gastrointestinal tissue and also fetal tissue. With regard to apoptosis there has been noticed that the level of the pro-apoptotic factor Bas (Bcl-2-associated X protein) was increased and the level of Bcl-xl, an anti-apoptotic factor, was decreased in human chrondocytes (cartilage cells). When exposed to T-2 mycotoxin. Furthermore, the level of Fas, an apoptosis-related cell-surface antigen and p53, a protein regulating the cell cycle, were increased.

 

Simplified biosynthesis of the T-2 Mycotoxin in F. sporotrichioides

Synthesis

T-2 mycotoxin is produced naturally by Fusarium fungi of which the most important species are: F. sporotrichioides, F. langsethiae, F. acuminatum and F. poae. These fungi are found in grains such as barley, wheat and oats. The production of this compound for research and commercial purposes is generally accomplished by cultivating some strain of T-2 mycotoxin producing fungi on agar plates. On these agar plates the fungi appear powdery and can yield substantial amounts of T-2 mycotoxin. For the isolation of the compound high pressure liquid chromatography is commonly used (HPLC).[16]

 

In the Fusarium species, biosynthesis of the T-2 mycotoxin often starts with trichodiene, and many of the species share a common route of oxidizations and cyclizations. As an example, from the F. sporotrichioides species, the important oxidation steps that occur start from trichodiene and goes to isotrichodiol. From there, the eleventh carbon atom is oxidized to form isotrichotriol. The ninth carbon is then oxidized, and trichotriol is formed, which then cyclizes to make isotrichodermol. After that, the fifteenth carbon is oxidized to form didecalonectrin, which leads to the fourth carbon being oxidized, and diacetoxyscirpenol is formed. The second to last step is the oxidation of the eighth carbon to make neosolaniol, which then undergoes slight modification to create the T-2 toxin.[17]

 

Toxicity

ADME properties

See also: ADME

Absorption and exposure

Humans and animals are generally exposed to T-2 mycotoxins through food. Certain grains can contain the toxin which makes it a threat to human health and an economic burden.[18] Unlike most biological toxins T-2 mycotoxin can be absorbed through intact skin. The compound can be delivered via food, water, droplets, aerosols and smoke from various dispersal systems. This makes it a potential biological weapon, however large amounts of the compound are required for a lethal dose. T-2 mycotoxin has an LD50 of approximately 1 milligram per kilogram of body weight.

 

The EFSA estimates that the mean exposure of T-2 in the EU lies between 12 and 43 ng/kg bw/day.[19] This range is below the TDI of 100 ng/ kg body weight for the sum of HT-2 and T-2 toxins which is used by the EFSA.

 

Distribution

T-2 mycotoxin is distributed uniformly throughout the body without preference to a specific organ or site. In rodents, plasma concentration levels peak around roughly thirty minutes after exposure, and in one study, the half-life of the T-2 toxin was seen to be less than twenty minutes. In a different study involving pigs, the distribution after four hours of IV injection was seen to be 15โ€“24% in the GI tract and 4.7โ€“5.2% in various other tissues.[20]

Anonymous ID: 30f01b June 3, 2025, 4:12 p.m. No.23118749   ๐Ÿ—„๏ธ.is ๐Ÿ”—kun   >>8758

>>23118735

Yes, which brings the question; why China buying farmland near military bases,

and we just saw Ukraine what a covert drone attack is capable of.

People would be dropping without knowing the cause.

Global panic would ensue.

Everyone would get "vaccinated" against the new pandemic 'Virus", just to be sure.

Mass casualty would be blamed on the 'virus'.