Anonymous ID: d102ba Dec. 19, 2018, 10:27 p.m. No.4387729   🗄️.is 🔗kun   >>7913

>>4387613

>>4387567

>Don't know how RX opioids got lumped in with them.

 

Ask Eric Holder that question. Largely unknown by the public, he played a huge role in creating the current problem of RX opiate dependence among the middle class, who are now willing consumers of street opioids.

 

excerpt:

"Drug companies benefit from a proverbial "revolving door" between government and special interests. A lawsuit in West Virginia was resolved in 2004 when Purdue Pharma agreed to pay $10 million over four years into drug abuse and education programs in the state. The settlement was negotiated on Purdue Pharma’s behalf by Eric Holder Jr., then a private attorney, who five years later became the chief of the Justice Department, which oversees DEA."

 

https://www.salon.com/2011/10/11/as_abuse_mounted_dea_boosted_painkiller_supply/#comments

Anonymous ID: d102ba Dec. 19, 2018, 11:08 p.m. No.4388096   🗄️.is 🔗kun   >>8177 >>8178 >>8229

>>4387913

 

sorry to hear that anon…

 

Here is some useful info:

 

1) Look into taking boron, magnesium, and vitamin D3 supplements for your arthritis

 

2) Kratom (mitragyna specieosa) which is a natural atypical opiate still legal online here in the US which aids with pain relief while mitigating Rx opioid addiction

 

3) BREAKING OUT OF THE VICODIN PRISON

There is also a biochemical story here that needs to be told and it has to do with an over the counter product that we have always been told is completely safe by the medical establishment (probably ever since we had our first fever as children). Tylenol, otherwise known as acetaminophen is a product that should have been removed from the market decades ago. Why you ask? Because it is toxic to both the brain and liver. In fact, it is the leading cause of acute liver failure world wide. Acute liver failure related to use of the painkiller accounted for 28% of all cases in 1998, and 51% in 2003.

 

Acetaminophen metabolites are toxic to both the liver and the brain by depleting intracellular glutathione. While BIG PHARMA rakes in 2 billion in annual sales WW, that is likely merely a drop in the bucket compared to the undocumented ROI that acetaminophen creates in profit from the various disorders and disease caused by it depleting the body's cellular stores of glutathione.

 

ACETAMINOPHEN should have NEVER BEEN COMBINED with OPIATES

Why? Because opiates block the cellular uptake of cysteine, which is necessary for glutathione production. Thus, drugs like Vicodin and Norco are pharmacological gulags, making the chronic user hypersensitive to pain due to the combined state of glutathione depletion and tolerance to opiates. Opiates are also pro-inflammatory antigens of the innate immune system.

https://en.wikipedia.org/wiki/Hydrocodone/paracetamol

 

If you are currently dependent on one of these opiate/acetaminophen "medications", you can take N-acetyl-cysteine (1,200 mg/day), which by passes the opiate block on cellular cysteine uptake. This will help you restore depleted glutathione levels and help you wean off these drugs.

N-acetyl-cysteine (NAC) is the anti-dote used in hospitals to rapidly reverse acute liver failure and is available as an inexpensive over-the-counter dietary supplement in the US. The reason why N-acetyl-cysteine is the first line treatment to reverse liver toxicity in the emergency room is because the acetyl group allows diffusion across cell membrane without the need of the transporter. Opiate addiction and the hypersensitivity to pain upon withdrawal of opiates is due in large part to being trapped in a state of chronic glutathione deficiency.

Anonymous ID: d102ba Dec. 19, 2018, 11:28 p.m. No.4388228   🗄️.is 🔗kun   >>8244

>>4388177

>>4388177

>I'll research the info you just gave and thanks.

 

The paper explains the biochemistry of how opiates block glutathione synthesis. Unfortunately, there are no definitive review papers which connects all the dots explaining why opiates with acetaminophen is a very bad combination.