Anonymous ID: 294562 Jan. 10, 2019, 9:07 a.m. No.4695474   🗄️.is 🔗kun

Secretary Pompeo

Verified account

 

@SecPompeo

4m4 minutes ago

More

American troops are coming home from Syria, but this isn’t a change of mission. We remain committed to the complete dismantling of the ISIS threat and the ongoing fight against radical Islamist terror. We are looking to our partners to do more going forward.

Anonymous ID: 294562 Jan. 10, 2019, 9:08 a.m. No.4695488   🗄️.is 🔗kun   >>5508 >>5524

ALERT: Chocolates, Candy Might Be Contaminated With Hepatitis A, FDA Warns

 

Candy?

 

https://breaking911.com/alert-chocolates-candy-might-be-contaminated-with-hepatitis-a-fda-warns/

Anonymous ID: 294562 Jan. 10, 2019, 9:10 a.m. No.4695502   🗄️.is 🔗kun   >>5523

https://www.nih.gov/news-events/news-releases/ai-approach-outperformed-human-experts-identifying-cervical-precancer?utm_source=TWITTER&utm_medium=TWITTER&utm_content=100000430419063&utm_campaign=Engagement&linkId=100000004619419

Anonymous ID: 294562 Jan. 10, 2019, 9:15 a.m. No.4695572   🗄️.is 🔗kun

>>4695549

Cyanide toxicity: Dx

Definition

Cyanide toxicity causes uncoupling of oxidative phosphorylation, interrupting aerobic metabolism and forcing a shift to anaerobic metabolism, leading to decreased O2 consumption, elevated lactate and severe metabolic acidosis.

 

Cyanide enters the body as gaseous HCN, water-soluble K-CN and Na-CN, and poorly soluble salts with Hg, Cu, Au, and Ag. Cyanogens, such as cyanogen-Br or –Cl, nitriles, and sodium nitroprusside may be converted to CN inside the body. Iatrogenic CN poisoning may be produced by high-dose or long-tern Na-nitroprusside IV therapy (>10 mcg/kg/min). CRF, pediatric patients and those with low thiosulfate reserves (POST-OP) are at increased risk of developing symptoms even at therapeutic doses.

 

With attention to the routes of exposure, a patient who presents with generalized weakness, malaise, collapse, neurologic symptoms, GI symptoms, and cardiopulmonary symptoms, CN poisoning must be high on the differential. The differential for these symptoms is broad (ACS, anaphylaxis, angina, MI, apnea, gastroenteritis, HA, meningitis, encephalitis, tachycardia, Hemlock poisoning, PE, cardiogenic shock, ischemic stroke, CO toxicity, hydrogen sulfide toxicity, Fe toxicity, INH toxicity, NSAID toxicity, azide toxicity, methanol toxicity, strychnine toxicity) and in addition to any known history, physical exam may reveal variable vitals, confusion, tachypnea, soot in the mouth and nose (following fire exposure) cherry-red skin, bright red retinal arteries and veins, and the smell of bitter almonds on the breath. Monitoring reveals a falsely reassuring SpO2, as no oxygen is being consumed by the cells hemoglobin remains fully saturated; supraventricular arrhythmias are common.

 

Labs:

 

Lactate: high

ABG: normal O2; metabolic acidosis may be masked by respiratory compensation

VBG: abnormally high O2, similar to ABG

Carboxyhemoglobin level may be checked to rule out co toxicity.

 

Toxicology screen for methanol, ethylene glycol, FE, ketones, salicylates to rule out confounders

 

*CN levels may be drawn but are not clinically useful for dx; typically not available in time to aid in diagnosis, may be useful to monitor treatment progress over a period of days

Anonymous ID: 294562 Jan. 10, 2019, 9:52 a.m. No.4696045   🗄️.is 🔗kun

do we have markers for precise dates in regards to evolving events like exact date of nat emergency declaration?