>>533191
>So is this the reason that Acetaminophen (Tylenol, Paracetamol) was invented? To get Aspirin out of circulation as "old-fashioned"?
>>533619
>news that aspirin caused Reye's Syndrome in kids
YES AND YES
DO I HAVE DIRECT PROOF? NO
BUT one thing is very clear. Acetaminophen IS NOT SAFE AND SHOULD NOT BE ON THE MARKET. THERE HAVE BEEN SEVERAL REVIEW PAPERS PUBLISHED ON THIS SUBJECT. (BTW, it was likely what really probably killed Prince).
Acetaminophen (Tylenol) should have been removed from the market decades ago due to its poor efficacy and because it is the number one cause of liver failure world wide and has been linked to autism and other neurodegenerative disorders (see graphic). Also, recently I learned that acetaminophen is being combined with DL-methionine in formulations used in 3rd world countries as an OTC pain reliever.
Methionine in paracetamol tablets, a tool to reduce paracetamol toxicity. Int. J Clin Pharmacol Ther Toxicol, 23(9), 497-500.
This is criminal in my opinion. The appropriate protective agent would be N-acetyl-cysteine, which is the antidote used in hospitals to reverse liver failure (NOT methionine!). Also methionine chelates mercury, which makes it far more bio available and toxic. I have no evidence that this combination was designed as a covert depopulation weapon, but if the combo is still being deployed IN 3RD WORLD COUNTRIES in light of the study below, then that would seem the most logical conclusion.
Depletion in Serum Levels of Folic Acid, Antioxidant Vitamins and Trace Elements in Female Wistar Rats Treated with Sub-toxic Dose of Acetaminophen/Methionine Combination – A Chronic Study
British Journal of Pharmaceutical Research, ISSN: 2231-2919,Vol.: 2, Issue.: 2 (April-June)
Context: Studies have identified that concurrent administration of methionine and acetaminophen (paracetamol) prevents tissue damage and both methionine and acetaminophen at high doses can induce oxidative stress. Antioxidants mediate against oxidative stress. Moreover, folic acid depletion has been identified to cause neural tube defects in neonates of affected female subjects.
Objective: The aim of this study is to investigate the impact of chronic exposure to sub-toxic dose of acetaminophen/methionine (ratio 5:1) on female Wistar rats, with emphasis on folic acid and antioxidant vitamins and minerals.
Material and Methods: Rats were divided into 3 groups with each group consisting of 8 rats and treated with acetaminophen/methionine, acetaminophen or saline daily by gastric gavage. The study lasted 30 days after which blood was obtained through retro-orbital bleeding.
Results: Results show that Wistar rats administered with 350 mg/kg BW (sub-toxic dose) of acetaminophen exhibited significant alteration (p<0.05) in levels of all trace elements (except Se) as well as vitamins (except vitamin A). Significant alterations in the levels of all vitamins (except riboflavin) and all minerals (except Cu, Mn, Se) (p<0.05) were also recorded in serum of rats administered with acetaminophen/methionine combination.
Discussion and Conclusion: Results of this study therefore suggest that chronic abuse of subtoxic dose of acetaminophen/methionine combination may induce alterations in levels of vital molecules, a situation which may increase an individual’s risk to oxidative stress-induced diseases and her neonate to neural tube defects.