Anonymous ID: 2421c9 March 3, 2020, 2:40 a.m. No.8306988   🗄️.is 🔗kun   >>6996 >>7077 >>7201 >>7260 >>7514 >>7595 >>7624

>>8306943

>Is COVID-19 a PURELY NATURAL zoonotic disease agent?

That's what the smart people think. Of course, arguments for why is is probably NOT a bioweapon never get any attention. Repost from a few days ago:

 

>>8282046 (PB)

Paper on a genetic analysis of the SARS-Cov-2 virus and it's origins. Very heavy and technical. It notably directly addresses the possibility of genetic engineering. Maybe it's correct. Maybe it's disinfo and damage control. Make of it what you will.

 

The Proximal Origin of SARS-CoV-2

http://virological.org/t/the-proximal-origin-of-sars-cov-2/398

 

Since the first reports of a novel pneumonia (COVID-19) in Wuhan city, Hubei province, China there has been considerable discussion and uncertainty over the origin of the causative virus, SARS-CoV-2. Infections with SARS-CoV-2 are now widespread in China, with cases in every province. As of 14 February 2020, 64,473 such cases have been confirmed, with 1,384 deaths attributed to the virus. These official case numbers are likely an underestimate because of limited reporting of mild and asymptomatic cases, and the virus is clearly capable of efficient human-to-human transmission. Based on the possibility of spread to countries with weaker healthcare systems, the World Health Organization has declared the COVID-19 outbreak a Public Health Emergency of International Concern (PHEIC). There are currently neither vaccines nor specific treatments for this disease.

SARS-CoV-2 is the seventh member of the Coronaviridae known to infect humans. Three of these viruses, SARS CoV-1, MERS, and SARS-CoV-2, can cause severe disease; four, HKU1, NL63, OC43 and 229E, are associated with mild respiratory symptoms. Herein, we review what can be deduced about the origin and early evolution of SARS-CoV-2 from the comparative analysis of available genome sequence data. In particular, we offer a perspective on the notable features in the SARS-CoV-2 genome and discuss scenarios by which these features could have arisen. Importantly, this analysis provides evidence that SARS-CoV-2 is not a laboratory construct nor a purposefully manipulated virus.

The genomic comparison of both alpha- and betacoronaviruses (family Coronaviridae ) described below identifies two notable features of the SARS-CoV-2 genome: (i) based on structural modelling and early biochemical experiments, SARS-CoV-2 appears to be optimized for binding to the human ACE2 receptor; (ii) the highly variable spike (S) protein of SARS-CoV-2 has a polybasic (furin) cleavage site at the S1 and S2 boundary via the insertion of twelve nucleotides. Additionally, this event led to the acquisition of three predicted O-linked glycans around the polybasic cleavage site.

Anonymous ID: 2421c9 March 3, 2020, 2:50 a.m. No.8307020   🗄️.is 🔗kun   >>7030 >>7042

>>8306996

The Canadian lab thing was a red herring. China already has all the SARS samples they could ever need (it started in China), plus thousands of other coronavirus strains literally flying around in their own backyards. They never had any need to steal anything.

 

>Gain of function research

What exactly is meant by that? In this case it means that it gained the ability to attach to a new receptor. Well, that what viruses naturally evolve to do. It's not a sign of unnatural modification.

Anonymous ID: 2421c9 March 3, 2020, 3:14 a.m. No.8307087   🗄️.is 🔗kun

>>8307071

>severity of Wuhan disease presentation is due to PRIOR exposure in a vaccine campaign?

No. It's probably due to the poor lung health of Chinese city dwellers (bad pollution, highest smoking rates in the world) and because the virus hadn't had time to evolve to be less virulent. Cold viruses do better when they don't make their hosts too ill to go out and spread it to other potential hosts. The people in Wuhan got it more directly from the source. The more time goes by, the more it will become like the common cold.