Anonymous ID: 39426f March 10, 2020, 7:40 a.m. No.8367350   🗄️.is 🔗kun   >>7479

>>8367282

>"scientists unsure"

 

The Media and WHO are pushing back saying its false hope, but we know they have an agenda. Read the article, not the headline.

 

This is common sense anyway. Bottom line, viruses are heat sensitive. After all, that is why the fuck we have fevers in the first place - to kill the virus.

 

But anyone can look at the map in real time. Its not spreading in areas near the equator and the death rate where the death rate is very low.

 

https://www.arcgis.com/apps/opsdashboard/index.html#/bda7594740fd40299423467b48e9ecf6

 

from the article:

”The virus is highly sensitive to high temperature”

 

The Guangzhou team based their study on every novel coronavirus case confirmed around the world between January 20 and February 4, including in more than 400 Chinese cities and regions. These were then modeled against official meteorological data for January from across China and the capital cities of each country affected.The analysis indicated that case numbers rose in line with average temperatures up to a peak of 8.72 degrees Celsius and then declined.

 

“Temperature … has an impact on people’s living environments … [and] could play a significant role in public health in terms of epidemic development and control,” the study said.It said also that climate may have played a part in why the virus broke out in Wuhan, the central China city where it was first detected.The virus that causes Covid-19 may have a temperature sweet spot at which it spreads fastest, a new study has suggested, but experts say people should avoid falling into the trap of thinking it will react to seasonal changes in exactly the same way as other pathogens, like those that cause the common cold or influenza.

 

The study, by a team from Sun Yat-sen University in Guangzhou, the capital of south China’s Guangdong province, sought to determine how the spread of the new coronavirus might be affected by changes in season and temperature.Published last month, though yet to be peer-reviewed, the report suggested heat had a significant role to play in how the virus behaves.

 

“Temperature could significantly change Covid-19 transmission,” it said. “And there might be a best temperature for viral transmission.”The virus is highly sensitive to high temperature”, which could prevent it from spreading in warmer countries, while the opposite appeared to be true in colder climes, the study said.

Anonymous ID: 39426f March 10, 2020, 8:08 a.m. No.8367533   🗄️.is 🔗kun

>>8367234

 

Hypothesis: Telmisartan should significantly reduce risk of CV infection.

 

Angiotensin II type 1 receptor is critical for CV viral cell entry and thus, infection in the lungs.

https://link.springer.com/article/10.1007/s00134-020-05985-9

 

If you are hypertensive, then you’re ACE II receptors are likely up regulated which places you at higher risk of both infection and rapid worsening.

https://www.bloomberg.com/news/articles/2020-03-09/top-virus-doctor-says-high-blood-pressure-is-major-death-risk

 

But, if you take Telmisartan, it works by down-regulating angiotensin II type 1 receptors through activation of peroxisome proliferator-activated receptor γ (anti-aging pathway).

 

Thus, drugs that down-regulate angiotensin II type 1 receptors should significantly reduce risk of infection.

 

NOTE: not all anti-hypertensives work this way

 

"Telmisartan decreased the expression of AT1R at the mRNA and protein levels in a dose- and time-dependent manner. Decreased AT1R promoter activity with unchanged mRNA stability suggested that telmisartan suppressed AT1R gene expression at the transcriptional level. However, the expression of AT1R was not suppressed by other AT1R antagonists such as candesartan or olmesartan."

 

https://academic.oup.com/cardiovascres/article/72/1/184/296597