antibiotics are basically all derived from mold, they are mycotoxins that inhibit bacterial growth, with a side effect of being immunosuppressive.
Azithromycin is very long lasting and prevents the expression of certain kinds of macrophages (CD4 iirc). That an "antibiotic" is being used effectively does not indicate that there is a "bacterial cause"; imho the immunosuppressive effect of the azithromycin is helping prevent the cytokine storm.
all of this hairsplitting about viral vs. bacterial vs plasmid is a moot point in light of what is known (and suppressed) about cell wall deficient organisms, check out the literature of Lida Mattman, read about Koch's Postulate.
What the COVID-19 does is attacks the surface proteins on the red blood cell and release the iron molecules that normally carry oxygen for the cell. when the iron is released it carries an electrical charge that causes tissue damage in the lungs (and possibly elsewhere?) so there is basically chemical lung damage, but the ventilators don't help because even with overpressure the red blood cells can't carry the oxygen. and the overpressure can cause injury itself.