>>9041471 (pb)

>>9041423

So it's just another form of SARS.

Severe Acute Respiratory Illness in the 21st century was first detected in 2002 and was called SARS-CoV-1. During this self-limited epidemic, there were 8,098 reported cases of SARS and 774 deaths.

In 2003, Chinese scientists identified that a virus nearly identical to SARS-CoV-1 was present in raccoons, dogs, and civets (wild cats) that was sold for food. Forty percent of animal traders and 33% of workers who slaughtered the animals had antibodies to the SARS virus. Sales of these animals were stopped by the government. The death rate was 10%.

In 2004, there was another smaller SARS outbreak linked to a medical laboratory in China. It was thought to have been the result of someone coming into direct contact with a sample of the SARS virus, rather than being caused by animal-to-human or human-to-human transmission.

>>9041736

The animals were used for food, and are still used for fur, as pets and hunting game. They are bred on the farms in terrible conditions and are subject to animal rights activists’ attempts of protection.

In 2012, MERS CoV (Middle East Respiratory Syndrome) is caused by a virus (more specifically, a coronavirus). Most MERS patients developed severe respiratory illness with symptoms of fever, cough, and shortness of breath. About 3 or 4 out of every 10 patients reported with MERS have died.

The outbreak was traced to a 46-year-old Yemeni man who, after arriving at the hospital by ambulance, spent 14 hours in the emergency department. His symptoms included coughing and shortness of breath, and the doctors noted that he had diarrhea for the two previous days. Blood tests initially indicated that he was suffering from kidney failure.

In 2015, several US laboratories, “utilizing the SARS-CoV infectious clone, generated and characterized a chimeric virus expressing the spike of bat coronavirus SHC014 in a mouse-adapted SARS-CoV backbone. The results indicate that group 2b viruses encoding the SHC014 spike in a wild type backbone can efficiently utilize multiple ACE II (angiotensin receptor two) receptor orthologs, replicate efficiently in primary human airway cells and achieve in vitro titers equivalent to epidemic strains of SARS-CoV.”

(Nature Medicine, published online November 9, 2015.)

>>9041758

It is said that SARS-CoV-2 originated in China when a cluster of families developed pneumonia cases on December 12, 2019. Chinese scientists rapidly isolated and sequenced the genome of the virus. The transmission was identified as person-to-person. Many animals may carry the virus but those are not identical to the human virus.

The virus uses ACE 2 (angioconverting enzyme 2) located in the CILIA of the lungs to attach in the proximity of the blood vessels. (Cilia are little appendages that stick out from cells. They whip back and forth and help cells move around in cellular fluids. They also help particles move past the cell.) This begins the cascade of "CYTOPATHIC" (cell destruction) effects and cytokine storm (any of a number of substances, such as interferon, interleukin, and growth factors, which are secreted by certain cells of the immune system and have an effect on other cells that are released and rapidly cause a severe sustained inflammatory response.

>>9041796

Dallas becoming a mini "hot zone" with highest single-day number of new cases 2 days in a row.

Bill Hemmer Reports May 5, 2020

>>9041851

Yeppers. Question is: was it deliberate?

Remember the headline: Coronavirus spread by woman using dry ice in Wuhan.

>>9041931

cap of article in Laura's tweet

The Effect of Chloroquine, Hydroxychloroquine and Azithromycin on the Corrected QT Interval in Patients with SARS-CoV-2 Infection

Originally published 29 Apr 2020

https://doi.org/10.1161/CIRCEP.120.008662

Abstract

Background - The novel SARs-CoV-2 coronavirus is responsible for the global COVID-19 pandemic. Small studies have shown a potential benefit of chloroquine/hydroxychloroquine ± azithromycin for the treatment of COVID-19. Use of these medications alone, or in combination, can lead to a prolongation of the QT interval, possibly increasing the risk of Torsade de pointes (TdP) and sudden cardiac death.

Methods - Hospitalized patients treated with chloroquine/hydroxychloroquine ± azithromycin from March 1st through the 23rd at three hospitals within the Northwell Health system were included in this prospective, observational study. Serial assessments of the QT interval were performed. The primary outcome was QT prolongation resulting in TdP. Secondary outcomes included QT prolongation, the need to prematurely discontinue any of the medications due to QT prolongation and arrhythmogenic death.

Results - Two hundred one patients were treated for COVID-19 with chloroquine/hydroxychloroquine. Ten patients (5.0%) received chloroquine, 191 (95.0%) received hydroxychloroquine and 119 (59.2%) also received azithromycin. The primary outcome of TdP was not observed in the entire population. Baseline QTc intervals did not differ between patients treated with chloroquine/hydroxychloroquine (monotherapy group) vs. those treated with combination group (chloroquine/hydroxychloroquine and azithromycin) (440.6 ± 24.9 ms vs. 439.9 ± 24.7 ms, p =0.834). The maximum QTc during treatment was significantly longer in the combination group vs the monotherapy group (470.4 ± 45.0 ms vs. 453.3 ± 37.0 ms, p = 0.004). Seven patients (3.5%) required discontinuation of these medications due to QTc prolongation. No arrhythmogenic deaths were reported.

Conclusions - In the largest reported cohort of COVID-19 patients to date treated with chloroquine/hydroxychloroquine {plus minus} azithromycin, no instances of TdP or arrhythmogenic death were reported. Although use of these medications resulted in QT prolongation, clinicians seldomly needed to discontinue therapy. Further study of the need for QT interval monitoring is needed before final recommendations can be made.

https://www.ahajournals.org/doi/10.1161/CIRCEP.120.008662

>>9042058

A lot of tests given (at least early on) were not specific for this strain, only that you have a cold-like virus. Remember, one-fifth of all common colds are coronaviruses. I heard a medical person (can't remember who but it was a Fox segment) say the test was for any COVID virus. This sort of slipped out during the interview and wasn't followed up, but I was thinking, wait, what? I think it had to do with the drive-through testing stations, which we don't hear anything about now, do we?