BOOMS INCOMING?
Been searching the meaning behind @POTUS's Orange and Black tie.
USDOT EXPLOSIVE PLACARDS are Orange with Black writing.
Possible Booms Incoming?
BOOMS INCOMING?
Been searching the meaning behind @POTUS's Orange and Black tie.
USDOT EXPLOSIVE PLACARDS are Orange with Black writing.
Possible Booms Incoming?
https://health.hawaii.gov/docd/disease_listing/kawasaki-syndrome/
About This Disease
Kawasaki syndrome (KS) is an acute febrile illness that primarily affects children younger than 5 years of age, and most commonly in children of Japanese or Korean descent.
Signs and Symptoms
Kawasaki syndrome typically occurs in three phases.
Phase 1 (acute): Most patients develop high, spiking fever for about 10 days with four or more of the following symptoms: body rash, conjunctivitis, sore throat, swollen lymph nodes mostly in the neck, reddish discolorations on the palms of the hands and the soles of the feet, and red, dry, cracked lips and extremely red swollen tongue (strawberry tongue). This phase is the most intense and symptoms can be the most severe.
Phase 2 (subacute phase): This phase begins as the fever and rash subsides with the tips of the fingers and toes begin to peel often. Patients may also develop joint pain, diarrhea, vomiting, and/or abdominal pain. This phase usually lasts about 2 weeks.
Phase 3 (convalescent phase): Clinical symptoms gradually fade. “Although KS is primarily self-limiting, the disease causes significant morbidity in most patients and can result in a range of cardiac and non-cardiac complications. Coronary artery abnormalities (CAA) can occur in more than 20% of untreated KS patients.”1 For a small percentage of children who develop heart problems, KS is fatal even with treatment.
Transmission
No one knows what causes Kawasaki Syndrome or how it’s spread. It doesn’t appear to be hereditary or contagious.
There is no firm evidence for person-to-person spread, although outbreaks of Kawasaki syndrome are consistent with an infectious etiology.
Diagnosis
There is no specific diagnostic tests for Kawasaki Syndrome. Diagnosis is based on clinical history and physical findings.
Treatment
Early diagnosis and treatment decreases the development of complications. The standard treatment is intravenous immunoglobulin and aspirin.
Immunity
There are no known measures that will prevent this illness.
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Abstract
Investigation of two outbreaks of Kawasaki syndrome (KS) in the United States in 1979 and in 1980 revealedno evidence of person-to-person transmission or of a common-source exposure among patients.Questionnaire data showed that KS was more likely to occur in children of middle and upper socioeconomic status than in those of lower status (P<0.05 and P<0.001 for the respective outbreaks) and that patients with KS had a higher incidence of an antecedent, primarily respiratory illness than did controls matched for age, sex, and race (83 per cent of patients in the first outbreak vs. 30 per cent of one control group, P<0.01, and vs. 36 per cent of another control group, P<0.02; and 56 per cent of patients in the second outbreak vs. 32 per cent of their controls, P<0.02). However, laboratory studies did not identify an etiologic agent for either KS or for the antecedent illness that may be a risk factor for KS. (N Engl J Med. 1981; 304:1568–75.)
https://health.hawaii.gov/docd/disease_listing/kawasaki-syndrome/
About This Disease
Kawasaki syndrome (KS) is an acute febrile illness that primarily affects children younger than 5 years of age, and most commonly in children of Japanese or Korean descent.
Signs and Symptoms
Kawasaki syndrome typically occurs in three phases.
Phase 1 (acute): Most patients develop high, spiking fever for about 10 days with four or more of the following symptoms: body rash, conjunctivitis, sore throat, swollen lymph nodes mostly in the neck, reddish discolorations on the palms of the hands and the soles of the feet, and red, dry, cracked lips and extremely red swollen tongue (strawberry tongue). This phase is the most intense and symptoms can be the most severe.
Phase 2 (subacute phase): This phase begins as the fever and rash subsides with the tips of the fingers and toes begin to peel often. Patients may also develop joint pain, diarrhea, vomiting, and/or abdominal pain. This phase usually lasts about 2 weeks.
Phase 3 (convalescent phase): Clinical symptoms gradually fade. “Although KS is primarily self-limiting, the disease causes significant morbidity in most patients and can result in a range of cardiac and non-cardiac complications. Coronary artery abnormalities (CAA) can occur in more than 20% of untreated KS patients.”1 For a small percentage of children who develop heart problems, KS is fatal even with treatment.
Transmission
No one knows what causes Kawasaki Syndrome or how it’s spread. It doesn’t appear to be hereditary or contagious.
There is no firm evidence for person-to-person spread, although outbreaks of Kawasaki syndrome are consistent with an infectious etiology.
Diagnosis
There is no specific diagnostic tests for Kawasaki Syndrome. Diagnosis is based on clinical history and physical findings.
Treatment
Early diagnosis and treatment decreases the development of complications. The standard treatment is intravenous immunoglobulin and aspirin.
Immunity
There are no known measures that will prevent this illness.
--------–
Abstract
Investigation of two outbreaks of Kawasaki syndrome (KS) in the United States in 1979 and in 1980 revealedno evidence of person-to-person transmission or of a common-source exposure among patients.Questionnaire data showed that KS was more likely to occur in children of middle and upper socioeconomic status than in those of lower status (P<0.05 and P<0.001 for the respective outbreaks) and that patients with KS had a higher incidence of an antecedent, primarily respiratory illness than did controls matched for age, sex, and race (83 per cent of patients in the first outbreak vs. 30 per cent of one control group, P<0.01, and vs. 36 per cent of another control group, P<0.02; and 56 per cent of patients in the second outbreak vs. 32 per cent of their controls, P<0.02). However, laboratory studies did not identify an etiologic agent for either KS or for the antecedent illness that may be a risk factor for KS. (N Engl J Med. 1981; 304:1568–75.)
https://www.nejm.org/doi/full/10.1056/NEJM198106253042603
If they are cashed/deposited by the person (dead person), that's a pretty big clue of nefarious activities.