The WE Forum and their RESET plans are connected to the company WE Work in New York, to the WE Charity in Canadaand the USA too, the WE-Tech Alliance in Canada, WE USA (Womens Enterprise) and other groups too. It is an entire network of CHANGE AGENTS.

As you may expect WE is a code that they use. It links them all to their inspiration, to the source of their Communist Utopian plan.

Note that Zamyatin's book was written in the Soviet Union, however it was published outside Russia in English before the original was ever published. This, to me, is a sign that it was intended as a propaganda piece to recruit people throughout the Capitalist world into the Communist International plans.

We (Russian: Мы, romanized: My) is a dystopian novel by Russian writer Yevgeny Zamyatin, written 1920–1921.[2] The novel was first published as an English translation by Gregory Zilboorg in 1924 by E. P. Dutton in New York. The novel describes a world of harmony and conformity within a united totalitarian state. It is believed that the novel had a huge influence on the works of Orwell and Huxley, as well as on the emergence of the genre of dystopia.[3] George Orwell claimed that Aldous Huxley's 1931 Brave New World must be partly derived from We, but Huxley denied it.

Attached is a PDF of the book to read, and to use in meme making, etc.

>>10171402

You should have taught her to scream 'I can't breathe and rip off the mask, and take some deep breaths of air.

Unlikely they would have forced her back in the mask. If they tried, she could have screamed again I can't breathe!, I need oxygen? Don't you have an oxygen mask?

>>10171456

Q has always made it clear that he is fake

Anyone who actually digs into hid drops knows that they are almost all LIES and GARBAGE.

He has been trying to teach us all to ACT FOR OURSELVES

To get off our asses and do things away from our keyboards.

Right from the beginning Anons have been exhorting us on this board to get out and talk to neighbors to form local MAGA action groups.

WE MUST RISE UP because NOBODY is going to save America

Only the joint action of We The People can do that.

Where We Go One We Go All

United Not Divided

>>10171406

A Closer Look At Ghislaine Maxwell's Relationship With Jeffery Epstein

Alana Goodman, co-author of "Convenient Death: The Mysterious Demise of Jeffrey Epstein" joined the Daily Caller's Stephanie Hamill to discuss the latest with Epstein's Alleged Madam Ghislaine Maxwell and to share her discoveries about Epstein while writing the book.

>>10171565

The Liberal Deep State loves people like you

They are all giggling right now as they sip their glasses of blood wine

They know that a citizen who is angry is wasting all his energy on feeding demons

Which weakens the citizen and makes him unable to protect himself when the Cabal comes calling

Puny weakling you are.

You have shriveled yourself through your anger

The wise man is cool and calm, and strikes the enemy at his weakest point when he least expects.

The wise man hasNO FEAR!

>>10171567

CNN hack John Berman clearly stated that in the Henry Ford studypatients were twice as likely to receive steroids which are proven to have a benefit.

This needs to be more widely know because Steroidal Anti-Inflammatory Drugs, such as Dexamethasone, do act against the Cytokine storm that causes swelling in the bronchial tubes and lungs leading to death. Cortico-Steroids like Prednisolone and Budesonide also prevent the symptoms that cause death.

There is much more to treatment of this disease than merely attacking the mechanism of viral production, i.e. Zinc and a Zinc Ionophore of some sort. HCQ is just one of many Zinc Ionophores that can be used.

It is pretty clear not that HCQ is NOT A MAGIC BULLET but it is Zinc which should be focused on. Once your cells are fortified with Zinc, respiratory viruses don't have much effect on you. Not flu, not the 200 or so Common Cold viruses, and maybe not even things like HIV.

>>10171832

Effects of Prednisolone Treatment on Cytokine Expression in Patients with Leprosy Type 1 Reactions

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1111887/

ABSTRACT

Leprosy type 1 reactions (T1R) are due to increased cell-mediated immunity and result in localized tissue damage. The anti-inflammatory drug prednisolone is used for treatment, but there is little good in vivo data on the molecular actions of prednisolone. We investigated the effect of prednisolone treatment on tumor necrosis factor alpha (TNF-α), interleukin-1β (IL-1β), IL-10, and transforming growth factor β1 (TGF-β1) mRNA and protein expression in blood and skin biopsies from 30 patients with T1R in India. After 1 month of prednisolone treatment the sizes of the skin granulomas were reduced, as were the grades of cells positive for TNF-α and IL-10 in skin lesions. Increased production of TGF-β1 was seen in skin lesions after 6 months of prednisolone treatment. Expression of mRNA for TNF-α, IL-1β, and TGF-β1 was reduced, whereas no change in IL-10 mRNA expression was detected during treatment. The circulating cytokine profiles were similar in patients with and without T1R, and prednisolone treatment had no detectable effects on cytokine expression in the blood. The data emphasize the compartmentalization of pathology in T1R and the importance of the immune response in the skin. Clinical improvement and cytokine expression were compared. Surprisingly, patients with improved skin and nerve function and patients with nonimproved skin and nerve function had similar cytokine profiles, suggesting that clinical improvement is not directly mediated by the cytokines studied here. This in vivo well-controlled study of the immunosuppressive effects of prednisolone showed that the drug does not switch off cytokine responses effectively.

= = = = = = = = = = = = = = = = = = = = =

Lupus and leprosy: beyond the coincidence

https://pubmed.ncbi.nlm.nih.gov/25412733/

Abstract

Systemic lupus erythematous (SLE) is an autoimmune disease that presents an increased susceptibility to infections which may trigger reactivation. Disease flares have been mostly associated with parvovirus B19, cytomegalovirus, EBV and Mycobacterium tuberculosis infections, but it is probable that many other agents may also induce innate and adaptive immune system stimulation including the production of autoantibodies as ANA, anti nDNA and anti-ß2-GPI mainly in lepromatous leprosy. Mycobacterium leprae not only may determine symptoms that mimic lupus flares, including autoantibodies production, but could also act as a trigger for lupus reactivation; however, its association is still not fully explored. As demonstrated for tuberculosis, it is quite possible that molecular mimicry may also be involved in the interface of these two diseases. Some studies reported shared epitopes among idiotypes derived from 8E7 and TH9 lepromatous antibodies and those obtained from SLE patients, and it could partially explain the triggering phenomenon of SLE caused by M. leprae. We report and discuss three Brazilian patients whose disease was inactive and presented disease flares concurrently with the diagnosis of leprosy.

The Lupus and Leprosy article is attached as PDF because it is stuck behind a paywall

>>10171701

This text version of the novel is the only one I can find on the net.

http://gutenberg.net.au/ebooks06/0608941.txt

This is odd because there seem to be lots of literary studies even though one analysis starts by saying that it is not a very good book. It's almost as if the establishment wants to guide your understanding of the book because something in it runs counter to their plans.