dChan - Q Origins Project Archive

REMEMBER: “COVID-19. There is an argument that it is ethnically targeted. COVID-19 attacks certain races disproportionately,” Kennedy said. “COVID-19 is targeted to attack Caucasians and black people. The people who are most immune are Ashkenazi Jews and Chinese.”

'a genetically engineered bioweapon that may have been “ethnically targeted” to spare Ashkenazi Jews and Chinese people'

This is what you get when you let the lowest IQ lunatics running the asslyum

https://nypost.com/2023/07/15/rfk-jr-says-covid-was-ethnically-targeted-to-spare-jews/

STATINS ARE POISON FOR PROFIT & FUNDING: STATIN PHARMAGEDDON

The term Pharmageddon almost appears to be created specifically for the marketing of statins.

You would die instantly without cholesterol – it is a fundamental part of every cell in your body.

Statin Drugs Scam: Side Effects include Rapid Aging, Brain Damage

FACT: Those with high levels of cholestrol live longer, those with low levels of cholestrol die earlier. Most heart attack patients' cholesterol levels did not indicate cardiac disease …

January 12, 2009. 5 min read. A new national study has shown that nearly 75 percent of patients hospitalized for a heart attack had cholesterol levels that would indicate they were not at high risk for a cardiovascular event, based on current national cholesterol guidelines

$29 Billion Reasons to Lie About Cholesterol: Making Profit by Turning Healthy People into Patients:

Big Pharma Suffers Major Blow: Study Says Treating High Cholesterol With Statins Is a Waste of Time & Money

In reality, research actually shows there is no relationship at all between blood cholesterol levels and the degree of atherosclerosis in your blood vessels.

STATINS ARE POISON…Get your loved one off of them!

PART TWO: Statins speed up the transition from "midlife vigor" to "debilitated old age'. Every cell in your body needs cholesterol!

Statin side effects mimic the effects of aging; they include fatigue, weakness, instability, memory loss, reduced mental capacity, dementia, neuropathy. slowed reactions, muscle atrophy (wasting), muscle pain, Myopathy, Rhabdomyolysis, Kidney failure, dyspnea(difficult or labored breathing) and heart failure, heart conditions, intestinal disease, pancreatic problems, diabetes, cataracts, nerve damage, reduced libido, depression, accidents, suicide and cancer…also linked to ALS Lou Gehrig's Disease and early onset Parkisons.

Every cell in our body needs cholesterol.

Recently scientists have found that statins interfere with our stem cells, which work to repair damage to our bodies and protect us from muscle and joint pain as well as memory loss.

A study from Tulane University found that statins prevented stem cells from performing their main functions, namely reproducing and replicating other cells in the body to carry out repairs; in particular, statins prevented stem cells from generating new bone and cartilage.3

According to Professor Reza Izadpanah, a stem cell biologist and lead author of the research, “Our study shows statins may speed up the aging process. . . People who use statins as a preventative medicine for health should think again as our research shows they may have general unwanted effects on the body which could include muscle pain, nerve problems and joint problems.”4

And to what purpose? The twenty-year Honolulu Heart Program study compared changes in cholesterol concentrations over twenty years with all-cause mortality. It found that those individuals who had low cholesterol levels were at increased risk of death.

Physicians’ Experiences as Patients with Statin Side Effects: A Case Series:

This is the first analysis to address the experience of physicians themselves affected by adverse effects (AEs) of statin medications, encompassing muscle, neuropathic, cognitive, and behavioral AEs.

The impact of statin AEs in physicians can be profound, professionally and personally, in some cases requiring major professional modification or early retirement.

Poor awareness of statin problems by medical providers, and low receptiveness to reports of such problems, can extend even to patients when they themselves are physicians.

Patient A: Atorvastatin 40 then 80 mg was followed by cognitive problems, neuropathy, and glucose intolerance in a Radiologist in his 50s

Patient B: Atorvastatin 10 mg was followed in 2 months by muscle weakness and myalgia in an Internist in his 40s

Patient C: Atorvastatin, ezetimibe/simvastatin, rosuvastatin at varying doses was followed shortly after by irritability, myalgia, and fatigue in a Cardiac Surgeon in his 40s

Patient Simvastatin 20 then 40 mg was followed in 4 years by mitochondriopathy, myopathy, neuropathy, and exercise intolerance in an Emergency Medicine physician in his 50s

Patient E: Simvastatin 20 mg and niacin 1000 mg was followed in one month by muscle weakness and myalgia in a Physical Medicine and Rehabilitation physician in his 50s

Patient F: Lovastatin 20 mg then simvastatin 20 mg, atorvastatin 20 mg, rosuvastatin 5 mg, niacin 20 mg and ezetimbe 10 mg was followed by muscle weakness and myalgia in an Obstetrician/Gynecologist in his 70s

Patient G: Ezetimibe/simvastatin and atorvastatin (dose unavailable) was followed shortly after by cognitive problems in a Radiologist in her 80s (probable causality).

Anonymous ID: a69e21 Nov. 10, 2023, 10:32 a.m. No.19894195 🗄️.is 🔗kun >>4201 >>4226

Dr. A, a radiologist in his 50s, stated that he developed marked cognitive problems and also neuropathy shortly after increasing his atorvastatin dose from 40 to 80 mg/day. Effects were new, marked, sustained, and interfered with his work, leading to difficulty understanding case presentations, to radiology reading errors (including right/left errors on readings), and to placement of orders for the wrong patients. These consequences to his professional accuracy led him to fear medicolegal repercussions. He did not share his cognitive concerns with his own physician, leery of risks and ramifications from such disclosure. However, he did share the neuropathy symptoms. His physician, whom Dr. A noted bore a “Top Doctor” designation, stated that no further assessment was warranted for neuropathy symptoms except in diabetics. No suggestion was made by his doctor of a possible statin role, or suggestion for a trial of statin discontinuation, until a medical student commented on the association of neuropathy to statin use.

Discontinuation of statins, after 3 years of sustained symptoms, led to rapid and striking improvement in cognitive function. By a week after discontinuation, Dr. A noted dramatic cognitive recovery, which he perceived to be approximately complete. He observed more gradual recovery of neuropathy symptoms, which he characterized as having improved by an estimated 95% at 8 months (improvement having apparently plateaued at that level).

Dr. D. experienced a clear decline in exercise tolerance that progressed over a 4 year period on statins, with development and progression of myalgias ultimately rated 8–9/10 in severity, and extreme lethargy. Discontinuation of statins led to gradual improvement in muscle pain: 3.5 years after statin discontinuation, he reports 75% improvement in myalgias, however, there has been no discernible improvement in exercise intolerance(2) Dr. D’s non-physician sister proposed the statin connection after she herself suffered intolerance. Dr. D’s physicians were generally dismissive of a statin connection. Dr. D describes a “pervasive skepticism” at the idea, with physicians “rolling their eyes” at the suggestion, even when he presented them with literature supporting the relationship.

Dr. D. described his own prior attitude toward statins as “elementary and naïve, that they were bad in some people, but that there would be clear manifestations and happen quickly within weeks. I thought a normal CK would essentially rule it out, that the symptoms were largely reversible.” He now emphasizes that the side effects “can be very disabling, can happen in substantially delayed fashion, the CK can be normal”

Dr. E tolerated simvastatin for 13 years, however after a 2 year trial off, he resumed simvastatin with niacin. He retired early to pursue athletic adventures, but a month after recommencing statin and niacin, developed new exercise intolerance, rapid muscle fatigue, and loss of muscle strength. He discontinued the statin a month later. Two years later, although notable improvement has occurred

Dr. F …atorvastatin was stopped in 2000 when his muscle weakness became severe, and improvement in walking was noted 2 months later, but rosuvastatin was prescribed due to elevated cholesterol levels, resulting in a rapid return of weakness. His cardiologist initially dismissed the possible connection of statins to his weakness, when the cardiologist’s own family member developed similar statin AEs, he began to investigate the relationship. Now, whenever he and his wife (also a physician) notice gait abnormalities in their friends and even strangers, they inquire whether or not they are on statins.

Dr. G developed confusion, disorientation, and short-term memory loss. She asked repetitive questions and had a short attention span. Statins were discontinued, followed by marked improvement in cognitive function. A month after discontinuation, she recalled running into one of her former colleagues in the grocery store and being able to immediately recognize the person (a clear improvement over her prior state). This acquaintance exclaimed that she looked much better than 8 months previously, when they had last interacted

Dr. G’s physician dismissed the potential statin connection. Her son stated, “He replied in a condescending tone of disbelief that I ‘read too much’”.

Dr. G. was referred to a neurologist, and treated with donepezil for a year under the assumption the cognitive problems may be Alzheimer’s. Other diagnoses that had been considered included depression and pseudo-dementia.

Anonymous ID: a69e21 Nov. 10, 2023, 10:33 a.m. No.19894201 🗄️.is 🔗kun >>4226

2 physicians saw arrest of progression, and symptom improvement after discontinuing the statin, however neither returned fully to their previous health states.

These cases underscore that commonly reported statin AEs—here encompassing muscle [4], cognitive [8], fatigue [13], neuropathic [9], and behavioral [10]—also afflict high functioning professionals such as physicians, in whom these symptoms can have profound professional implications, contributing in some to early retirement or persistent disability.

These cases reprise a number of observations from other settings. Symptom onset can be delayed [4, 8]. Older age is a risk factor for statin AEs [5, 14, 15], and people continue to age while taking statins—moreover, physiological aging may be accelerated by the processes that underlie statin AEs [5]. Higher dose increases risk (Drs. A, C, D) [5]. For those who remain on statins after the first symptom arises, emergence of symptoms spanning several categories is not uncommon, likely reflecting common pathophysiological foundations [5].

Rarely, symptoms may initially worsen with discontinuation. (We note this is consistent with evidence that antioxidant effects can arise (and reverse) quickly, prior to lipid effects [16]; while some prooxidant effects, linked for instance to recovery of lipid transport of antioxidants, may take longer to reverse. This may, in some, engender an initial worsening of statin-induced prooxidant-antioxidant balance, on statin discontinuation.) Resolution of AEs can be incomplete (whether by patient report or objective testing) [6, 7] and residual disability may be profound [4, 17].

Statin AEs, including muscle wasting, weakness, and exercise intolerance as well as neuropathy perhaps in particular [18] (but also cognitive dysfunction) may fail to fully resolve clinically [19, 20] and pathologically [7]. (Indeed, there may be failure of full resolution pathologically even when there is apparent clinical resolution [19].) Persistent muscle problems can reflect mitochondrial myopathy [5–7]. Regarding cases in Table 2: Behavioral symptoms may be recognized by others rather than the individual in whom they occur (Dr. C) [21, 22]. Athletes may be at special risk for statin muscle problems (Dr. E) [23, 24]. Physicians seeing those who experience statin AEs may be dismissive of symptoms or of a possible statin connection—even, we find here, when reported by physicians-as-patients [3].

Physicians' Experiences as Patients with Statin Side Effects: A Case Series

https://pubmed.ncbi.nlm.nih.gov/28217821/

Anonymous ID: a69e21 Nov. 10, 2023, 10:38 a.m. No.19894226 🗄️.is 🔗kun >>4424

>>19894201

>>19894195

>>19894184

Physicians' Experiences as Patients with Statin Side Effects: A Case Series

https://pubmed.ncbi.nlm.nih.gov/28217821/